| slow maturing primary exo-erythrocytic schizonts of plasmodium berghei in an experimentally infected tree-rat (thamnomys surdaster). | | 1974 | 4607414 |
| an ultrastructural study of the pre-erythrocytic development of plasmodium berghei in the tree rat, thamnomys surdaster. | | 1972 | 5038729 |
| [another strain of babesia rodhaini in the african rodent thamnomys surdaster]. | | 1953 | 13124080 |
| tree rat, thamnomys surdaster surdaster, in laboratory research. | the breeding of successive generations of thamnomys in the laboratory made possible a study of its bionomics and an evaluation of its importance in parasitological and malarial research. | 1963 | 14075691 |
| plasmodium berghei: cyclical transmissions by experimentally infected anopheles quadrimaculatus. | a number of strains of plasmodium berghei were isolated from sporozoites of anopheles dureni. laboratory-bred anopheles quadrimaculatus fed on carriers of the newly isolated strains showed overwhelming midgut infections and moderate or mild salivary gland infections. successive cyclic transmissions by the bite of experimentally infected a. quadrimaculatus in laboratorybred tree rats (thamnomys surdaster) were carried out. | 1964 | 14169345 |
| a study of plasmodium berghei in thamnomys surdaster, and in other experimental hosts. | | 1964 | 14205883 |
| rodent malaria in the natural host--irradiated sporozoites of plasmodium berghei induce liver-stage specific immune responses in the natural host grammomys surdaster and protect immunized grammomys against p. berghei sporozoite challenge. | the choice of the host in studying host-parasite interactions is of crucial importance, and the use of a natural host is most appropriate in answering pertinent questions related to human malaria. the grammomys surdaster is the natural host and reservoir of the rodent malaria parasite plasmodium berghei. this natural host is difficult to protect by irradiated sporozoite immunization, a situation comparable to what has been observed in humans with p. falciparum. this is in contrast to the complet ... | 2001 | 14513935 |
| susceptibility of grammomys surdaster thicket rats to trypanosoma brucei gambiense infection. | human african trypanosomiasis is caused by trypanosoma brucei gambiense and t. b. rhodesiense. historically, a treatment relapse rate of about 5% is observed in patients treated with melarsoprol, an arsenical derivative used for treatment of both gambiense and rhodesiense second stage sleeping sickness. more recently, relapse rates up to 30% are noted in gambiense sleeping sickness foci in angola, sudan and uganda. therefore, who established a network on treatment failure and drug resistance in ... | 2005 | 16135191 |
| how reliable are models for malaria vaccine development? lessons from irradiated sporozoite immunizations. | models occupy a key position in the development of anti-parasitic vaccines, yet their relevance has been seldom addressed. it is customary to admit that malaria vaccine development requires easy-to-handle, laboratory models. animal models involving predominantly inbred rodents and primates as parasite hosts are currently the basic tools for the study of host-parasite interactions. literature however indicates that the induction of host protection is more difficult in natural host-parasite pairs ... | 2006 | 17102561 |
| isolation of trypanosoma brucei gambiense from cured and relapsed sleeping sickness patients and adaptation to laboratory mice. | sleeping sickness due to trypanosoma brucei (t.b.) gambiense is still a major public health problem in some central african countries. historically, relapse rates around 5% have been observed for treatment with melarsoprol, widely used to treat second stage patients. later, relapse rates of up to 50% have been recorded in some isolated foci in angola, sudan, uganda and democratic republic of the congo (drc). previous investigations are not conclusive on whether decreased sensitivity to melarsopr ... | 2011 | 21526217 |
| grammomys surdaster, the natural host for plasmodium berghei parasites, as a model to study whole-organism vaccines against malaria. | inbred mice are commonly used to test candidate malaria vaccines, but have been unreliable for predicting efficacy in humans. to establish a more rigorous animal model, we acquired african woodland thicket rats of the genus grammomys, the natural hosts for plasmodium berghei thicket rats were acquired and identified as grammomys surdaster by skull and teeth measurements and mitochondrial dna genotyping. herein, we demonstrate that thicket rats are highly susceptible to infection by p berghei, an ... | 2017 | 28115674 |
| [eimeria vinckei n. sp., intestinal parasite of thamnomys surdaster surdaster]. | | 1954 | 13218473 |
| [cordylobia ruandae n. sp., new fly with subcutaneous larva parasitizing the subcutaneous tissue of a rodent, grammomys surdaster, in ruanda-urundi, belgian congo]. | | 1953 | 13159020 |
| [observations on the biology of cordylobia ruandae and its host grammomys dolichurus surdaster in rwanda. parasitism of pupae of this fly by chalcidoides]. | | 1964 | 14323529 |
| [biology of grammomys surdaster thomas and wroughton in ruanda-urundi with relation to its parasite cordylobia ruanda fain, 1953]. | | 1956 | 13351171 |
| dynamics and outcomes of plasmodium infections in grammomys surdaster (grammomys dolichurus) thicket rats versus inbred mice. | investigations of malaria infection are often conducted by studying rodent plasmodium species in inbred laboratory mice, but the efficacy of vaccines or adjunctive therapies observed in these models often does not translate to protection in humans. this raises concerns that mouse malaria models do not recapitulate important features of human malaria infections. african woodland thicket rats (grammomys surdaster) are the natural host for the rodent malaria parasite plasmodium berghei and the susp ... | 2020 | 32815499 |