| assessment of occupational risk for hantavirus infection in arizona and new mexico. | differentiating occupational exposure from other potential domestic or recreational exposure(s) for sin nombre virus (snv) infection is an epidemiologic challenge. interviews on work-related activities were conducted, and serum specimens were obtained from 494 workers in arizona and new mexico. these workers may have been exposed to rodents and rodent excreta at work, but their primary occupation did not require rodent contact (national park service [n = 193]; navajo agricultural product industr ... | 1997 | 9172092 |
| serologic survey for hantavirus infection in domestic animals and coyotes from new mexico and northeastern arizona. | to determine whether animals had serologic evidence of infection with sin nombre virus (snv). | 1998 | 9540866 |
| hantavirus pulmonary syndrome--colorado and new mexico, 1998. | hantavirus pulmonary syndrome (hps) is a severe cardiopulmonary illness resulting in death in approximately 45% of cases. the most frequently recognized etiologic agent of hps in north america, sin nombre virus (snv), is transmitted to humans from its primary rodent reservoir, peromyscus maniculatus (deer mouse), by direct contact with infected rodents, rodent droppings, or nests or through inhalation of aerosolized virus particles from mouse urine and feces. sporadic cases occur throughout the ... | 1998 | 9639364 |
| implications of the analogy between recombinant cytokine toxicities and manifestations of hantavirus infections. | the etiologic hantavirus of the 1993 emergence of an acute pulmonary failure syndrome in the area around northwestern new mexico was quickly recognized as related to the hantaan virus responsible for the outbreak of korean epidemic hemorrhagic fever (ehf) among un troops in 1951. discovery of the new disease which was named the hantavirus pulmonary syndrome (hps) and its causative agent the sine nombre virus (snv) inspired detailed comparisons between the two disorders. major damage to the epith ... | 1998 | 10850356 |
| prevalence of antibodies to sin nombre virus in humans living in rural areas of southern new mexico and western texas. | | 2001 | 11226584 |
| neutralizing antibodies and sin nombre virus rna after recovery from hantavirus cardiopulmonary syndrome. | patients who later have a mild course of hantavirus cardiopulmonary syndrome (hcps) are more likely to exhibit a high titer of neutralizing antibodies against sin nombre virus (snv), the etiologic agent of hcps, at the time of hospital admission. because administering plasma from patients who have recovered from hcps to those in the early stages of disease may be an advantageous form of passive immunotherapy, we examined the neutralizing antibody titers of 21 patients who had recovered from snv ... | 2004 | 15109416 |
| hantavirus pulmonary syndrome--five states, 2006. | hantavirus pulmonary syndrome (hps) is a rodentborne viral disease characterized by severe pulmonary illness and a case-fatality ratio of 30%-40%. sin nombre virus causes the majority of hps cases in the united states, and the deer mouse (peromyscus maniculatus) is its predominant reservoir. this report describes an increase in human cases of hps reported during january-march 2006 from arizona, new mexico, north dakota, texas, and washington state. the findings emphasize the need for renewed att ... | 2006 | 16760891 |
| deer mouse movements in peridomestic and sylvan settings in relation to sin nombre virus antibody prevalence. | prevalence of antibody to sin nombre virus (snv) has been found to be nearly twice as high in deer mice (peromyscus maniculatus) in peridomestic settings as in sylvan settings in two studies in montana and one in new mexico. we investigated whether this difference may be related to a difference in deer mouse movements in the two settings. we used radiotelemetry to determine home range size and length of movement for 22 sylvan (1991-1992) and 40 peridomestic deer mice (1995-1999). we also determi ... | 2006 | 17255448 |
| genetic reassortment among viruses causing hantavirus pulmonary syndrome. | in order to determine the frequency and characteristics of reassortment among viruses causing hantavirus pulmonary syndrome (hps), mixed infections were initiated in tissue culture by using two closely related strains of sin nombre virus, cc107 (from eastern california) and nmr11 (from new mexico), which share the same species of rodent host in nature, the deer mouse (peromyscus maniculatus). potential reassortant virus plaques were screened by multiplex rt-pcr, using primers specific for indivi ... | 1998 | 9501041 |
| small mammal survival and trapability in mark-recapture monitoring programs for hantavirus. | following the 1993 hantavirus pulmonary syndrome (hps) epidemic in the south-western united states, mammalogists and epidemiologists instituted long-term studies to monitor population density and prevalence of infection in rodents which constitute the reservoir for sin nombre virus (snv). in this study, field techniques used in sampling small mammals for snv infection were evaluated to determine if trapping and handling protocols were having significant effects on future trapability or mortality ... | 1998 | 9476220 |
| mild hantaviral disease caused by sin nombre virus in a four-year-old child. | | 1995 | 8786902 |
| convalescent pulmonary dysfunction following hantavirus pulmonary syndrome in panama and the united states. | the objective of this study was to document persistent pulmonary symptoms and pulmonary function abnormalities in adults surviving hantavirus pulmonary syndrome (hps). acute infection by most hantaviruses result in mortality rates of 25-35%, while in panama the mortality rate of 10% is contrasted by an unusually high incidence. in all types of hps, the viral prodrome, cardiopulmonary phase due to massive pulmonary capillary leak syndrome, and spontaneous diuresis are followed by a convalescent p ... | 2010 | 20524006 |
| temporal and geographic evidence for evolution of sin nombre virus using molecular analyses of viral rna from colorado, new mexico and montana. | all viruses in the family bunyaviridae possess a tripartite genome, consisting of a small, a medium, and a large rna segment. bunyaviruses therefore possess considerable evolutionary potential, attributable to both intramolecular changes and to genome segment reassortment. hantaviruses (family bunyaviridae, genus hantavirus) are known to cause human hemorrhagic fever with renal syndrome or hantavirus pulmonary syndrome. the primary reservoir host of sin nombre virus is the deer mouse (peromyscus ... | 2009 | 19602267 |
| demographic factors associated with prevalence of antibody to sin nombre virus in deer mice in the western united states. | we used long-term data collected for up to 10 yr (1994-2004) at 23 trapping arrays (i.e., webs and grids) in arizona, colorado, montana, and new mexico to examine demographic factors known or suspected to be associated with risk of infection with sin nombre virus (snv) in its natural host, the deer mouse (peromyscus maniculatus). gender, age (mass), wounds or scars, season, and local relative population densities were statistically associated with the period prevalence of antibody (used as a mar ... | 2007 | 17347388 |