| non-ulcer dyspepsia and short term de-nol therapy: a placebo controlled trial with particular reference to the role of campylobacter pylori. | this double blind randomised study tested the effectiveness of colloidal bismuth subcitrate (de-nol) in non-ulcer dyspepsia (nud) and if any benefit is associated with clearance of campylobacter pylori (c pylori) from the gastric mucosa. sixty six patients with dyspepsic symptoms, normal abdominal ultrasound, and upper gi endoscopy, were randomly allocated to placebo or de-nol for eight weeks. antral biopsies were taken for bacteriological and histological examination, and endoscopies and clinic ... | 1988 | 3058555 |
| [campylobacter pyloridis and its relation to peptic ulcer and chronic gastritis]. | | 1988 | 3059039 |
| [campylobacter pylori and gastric ulcer]. | | 1988 | 3059629 |
| [campylobacter pylori]. | | 1988 | 3060270 |
| [bacteriologic, histologic and serologic studies of the detection of campylobacter pylori in stomach diseases]. | | 1988 | 3062918 |
| [campylobacter pylori. is a routine search for it useful?]. | | 1988 | 3063272 |
| [gastritis, peptic ulcer and campylobacter pylori]. | | 1988 | 3064995 |
| [campylobacter pylori and therapy with bismuth salts]. | | 1988 | 3067064 |
| [campylobacter pylori: a new light on the etiology of chronic gastritis and peptic ulcer?]. | | 1988 | 3067265 |
| campylobacter pylori infection and its relation to chronic gastritis. an endoscopic, bacteriologic, and histomorphologic study. | campylobacter pylori was isolated from 27 of 61 gastric antral biopsy specimens and from 8 of 61 duodenal biopsy specimens. a significant correlation between the occurrence of c. pylori and chronic active gastritis was demonstrated. however, the presence of the bacterium on normal mucosa weakens the theory of c. pylori as a primary causal organism. there was a significant correlation between isolation of c. pylori and erosive lesions in the antral mucosa as diagnosed by endoscopy. no correlation ... | 1988 | 3067331 |
| the protective role of gastric acid. | this paper reviews the relationship between gastric acid secretion and infection and the protective role of gastric acid as a primary bactericidal barrier and modulator of gastrin section. gastric acid is bactericidal at ph 3 or less, but reduction of acidity predisposes to infection with a wide variety of bacteria. bacterial infections or hyperpyrexia may be associated with a marked reduction in gastric acid secretion, and campylobacter pylori has been suggested as one cause of epidemic hypochl ... | 1988 | 3067334 |
| the campylobacter pylori story. | the campylobacter pylori story began before the turn of the century, with early works describing 'spirochaetes' in the gastric mucosa of animals. culture of the organism in 1982 enabled investigators to make sense of the many previous works concerning the microbiology, biochemistry, and histology of the gastric mucosa. whereas some physicians remain skeptical of c. pylori's pathogenic role, those who have studied the new organism believe it is a major gastrointestinal pathogen and see the possib ... | 1988 | 3067335 |
| [what is safe in the treatment of campylobacter pylori-induced gastritis and campylobacter pylori-associated peptic ulcer?]. | | 1988 | 3069787 |
| [the role of campylobacter pyloridis in the etiology and pathogenesis of peptic ulcer]. | | 1988 | 3070803 |
| [detection of campylobacter pylori in gastric biopsies: a comparative study of 4 histopathologic methods and scanning electron microscopy]. | | 1988 | 3071267 |
| does the choice of acute treatment influence future ulcer relapse? | despite the fact that the direct effect of drugs used for healing of ulcers does not last longer than hours, or at most a few days, some clinical studies have indicated that the rate of new ulcers after short courses of treatment depends on the drug used for healing. several studies have now shown that the development of new ulcers in the same, regardless whether active ulcers have been healed with antacids or histamine h2-receptor antagonists. this lack of difference in clinical outcome is very ... | 1988 | 3072664 |
| campylobacter pylori. | worldwide campylobacter pylori is a major cause of active chronic gastritis in man. this curved spiraled microorganism can readily be detected within the mucusgel especially in the antrum, in particular in patients suffering from peptic ulcer disease or non-ulcer dyspepsia, rarely in individuals with normal gastroduodenal mucosa. increasingly arguments are being presented in support of a pathogenetic role of c. pylori in non-ulcer dyspepsia and in peptic ulcer disease. there is a striking discor ... | 1988 | 3072666 |
| campylobacter pyloridis is not responsible for duodenal ulcer formation, result of a controlled therapeutic trial. | | 1988 | 3074085 |
| [campylobacter pylori and its role in the etiopathogenesis of gastroduodenal ulcer]. | | 1987 | 3078194 |
| campylobacter pyloridis and associated gastritis: investigator blind, placebo controlled trial of bismuth salicylate and erythromycin ethylsuccinate. | an investigator blind trial was performed comparing bismuth salicylate, erythromycin ethylsuccinate, and placebo in the treatment of campylobacter pyloridis associated gastritis in patients without peptic ulceration. fifty patients fulfilled the study criteria. there was a strong correlation between the presence of c pyloridis and histologically confirmed gastritis. clearance of organisms led to improvement of the gastritis. c pyloridis was cleared from 15 patients; of these, 13 had gastritis in ... | 1986 | 3092967 |
| campylobacter pyloridis: what role in gastritis and peptic ulcer. | | 1986 | 3094652 |
| antibodies to campylobacter pyloridis in household contacts of infected patients. | | 1987 | 3103831 |
| [isolation of campylobacter pyloridis from gastric mucosa of patients with gastric complaints and susceptibility of the isolates on antibiotics]. | | 1987 | 3119739 |
| campylobacter pylori, gastritis, and peptic ulcer disease in central africa. | | 1987 | 3120933 |
| campylobacter pylori in central africa. | | 1988 | 3122939 |
| campylobacter pylori: prospective analysis of clinical and histological factors associated with colonization of the upper gastrointestinal tract. | the association of campylobacter pylori (c.p.) colonization of the upper gastrointestinal tract with five predefined anamnestic variables, seven symptoms of dyspepsia, and various blindly evaluated histological criteria, was prospectively investigated in a consecutive series of 149 patients submitted to upper gastrointestinal tract endoscopy. colonization was determined by biopsy urease tests and histological searches. significant differences (p less than 0.05) between c.p.-positive and c.p.-neg ... | 1988 | 3133219 |
| evaluation of a new selective medium for campylobacter pylori. | contaminating bacteria from the oropharynx and bacteria that colonise the stomachs of patients with a high gastric ph impede the isolation of campylobacter pylori from gastric biopsy specimens. commercially available selective supplements are inhibitory to this organism and therefore a specific selective medium is needed for isolation. potential selective agents were evaluated for their activity against 97 strains of campylobacter pylori. a modification of skirrow's medium was developed; cefsulo ... | 1988 | 3141172 |
| enzyme-linked immunosorbent assay for detection of immunoglobulin a and g antibodies to campylobacter pylori. | an enzyme-linked immunosorbent assay (elisa) employing an acid-glycine extract was used to detect igg and iga antibodies to campylobacter pylori in sera from 179 patients with upper gastrointestinal disease, 174 blood donors and 65 children. the incidence of positive elisa results clearly increased with the severity of histopathologic findings in the antrum mucosa and was also high in patients with peptic ulcers and gastric cancer. the incidence in blood donors and children was much lower and in ... | 1988 | 3141173 |
| susceptibility of clinical isolates of campylobacter pylori to twenty-one antimicrobial agents. | the mics of 21 antimicrobial agents were determined for 97 clinical isolates of campylobacter pylori. the beta-lactams (penicillin, ampicillin, cefoxitin and cephalexin), macrolides (erythromycin and azithromycin), quinolones (ciprofloxacin and ofloxacin), nitrofurans, gentamicin and tetracycline all had mic90 values of less than or equal to 0.5 mg/l. aztreonam, flucloxacillin, amifloxacin and rifampicin had moderate activity. all isolates were resistant to vancomycin, cefsulodin and amphoterici ... | 1988 | 3141174 |
| comparison of different antigen preparations in an evaluation of the immune response to campylobacter pylori. | this study presents a novel approach to the analysis of protein antigens of campylobacter pylori for use in serology. protein fractions of this bacterium were resolved in polyacrylamide gel electrophoresis, eluted from gel strips in an electric field and used for coating of microtiter plates in an elisa-type assay run with a small set of sera from both infected and non-infected patients. reactivity and discriminative power of the different fractionated antigens (1-9) and crude antigen preparatio ... | 1988 | 3141175 |
| demonstration of a cytotoxin from campylobacter pylori. | in order to determine if campylobacter pylori produces a cytotoxin, a study was performed using bacterial lysates from three clinical isolates of the organism. the lysates were cytotoxic for chinese hamster ovary cells, as determined by a microtiter assay. the lysates were also lethal for mice after intraperitoneal injection. loss of toxicity and lethality followed trypsinization, heating and acidification of cell-free lysates. it is concluded that the toxic factor(s) in campylobacter pylori may ... | 1988 | 3141176 |
| in vitro activity of pentacaine against campylobacter pylori. | | 1988 | 3141177 |
| possible pathogenic mechanisms of campylobacter pylori. | | 1988 | 3141178 |
| risk factors for persistent diarrhoea. | with a systematically sampled population of children aged under 5 attending this centre for diarrhoeal disease research during 1983-5 a retrospective analysis of persistent diarrhoea (defined as greater than 14 days' duration) was performed to identify the possible risk factors for this syndrome. of the 4155 children included in the analysis, 410 (10%) gave a history of persistent diarrhoea. a comparison with children with acute diarrhoea matched for age showed that 11 factors were correlated wi ... | 1988 | 3142603 |
| campylobacter pylori in gastroduodenal disease. | | 1988 | 3144601 |
| [campylobacter pylori]. | | 1987 | 3149939 |
| [campylobacter pylori and gastroduodenal pathology]. | | 1987 | 3149940 |
| [campylobacter pylori: a retrospective study of 150 gastric biopsies with chronic type b gastritis]. | | 1987 | 3149942 |
| [gastroduodenal ulcer and campylobacter pylori. behavior under treatment with de-nol or metronidazole]. | | 1987 | 3149943 |
| [isolation of campylobacter pylori from the gastric mucosa by gram stain and culture]. | | 1988 | 3152671 |
| [frequency of campylobacter pylori in gastroduodenal lesions at an outpatient gastroenterology clinic]. | | 1988 | 3152672 |
| [gastritis in children associated with campylobacter pylori]. | | 1988 | 3152673 |
| campylobacter pyloridis--a stomach pathogen. | | 1986 | 3153584 |
| campylobacter pylori in gastroduodenal diseases: current views--future directions. proceedings of an international workshop. copenhagen, 15 and 16 october 1987. | | 1988 | 3166526 |
| monotherapy or polychemotherapy in the treatment of campylobacter pylori-related gastroduodenal disease. | three pilot studies were performed to evaluate the efficacy of bismuth subsalicylate (bss) and nitrofurantoin to eradicate campylobacter pylori colonization in man. nitrofurantoin 3 x 100 mg capsules for 10 days did not clear c. pylori in any of 13 patients, and neither did the combination of bss and nitrofurantoin suspension (0/6 patients). immediately after high dose bss therapy 3 x 900 mg for 28 days, 8/17 patients (47%) had negative cultures and rapid urease tests. there was recrudescence in ... | 1988 | 3166527 |
| natural sources and microbiological characteristics of campylobacter pylori. | | 1988 | 3166529 |
| histological aspects of campylobacter pylori colonisation and infection of gastric and duodenal mucosa. | the main features of the histopathology of campylobacter pylori-associated gastritis are reviewed. the organism is strongly associated with the type b pattern of chronic gastritis. it colonizes the abnormal antrum and is not usually seen on the normal mucosa. however it fails to colonize the abnormal antrum in the type a gastritis of pernicious anaemia. when present in the duodenum it is in conjunction with severe duodenitis and gastric metaplasia. although the organisms are present in over 70% ... | 1988 | 3166530 |
| pathogenetic implications of ultrastructural findings in campylobacter pylori related gastroduodenal disease. | there is now substantial evidence that campylobacter pylori (cp) is able to colonize the gastroduodenal mucosa and is responsible for active chronic gastritis, its role in duodenitis, gastric ulceration and duodenal ulceration is still under debate. cp has a lot of characteristics which are prerequisites for a pathogen: the typical s-shape, the corkscrew-like movement and the powerful urease and protease enzymes. these features allow a rapid movement through the mucous layer to permit access to ... | 1988 | 3166531 |
| possible pathogenetic pathways of campylobacter pylori in gastro-duodenal disease. | the exact pathogenic mechanisms involved in campylobacter pylori associated gastro-duodenal inflammation are unclear. c. pylori adheres to gastric type epithelium and colonisation is associated with a number of ultrastructural abnormalities. other factors thought important include the marked bacterial urease activity, enzymatic degradation of the mucous layer and possible toxins. the induced inflammatory reaction will also contribute to tissue damage and a breakdown of normal defense mechanisms. | 1988 | 3166532 |
| immune response of the gastric mucosa to campylobacter pylori. | the histological features of non-autoimmune chronic gastritis are consistent with those of a local immune response by the gastric mucosa. if campylobacter pylori is causative in this condition, then the immune response will be in part specific for this organism. antibodies to c. pylori are present in the gastric juice of infected patients. coating of mucosal c. pylori by antibody in vivo can be demonstrated by immunohistochemistry, and a neutrophil infiltrate (active gastritis) is associated wit ... | 1988 | 3166533 |
| an enzyme-linked immunosorbent assay for the serodiagnosis of campylobacter pylori-associated gastritis. | an elisa system, using an acid extractable cell surface c. pylori antigen, has been used to detect anti-c. pylori antibodies in 176 endoscoped patients. the antibody concentration was related to histological diagnosis and colonisation with campylobacter-like organisms. the criteria for sero-positivity was determined from the median antibody concentration in a group of 368 non-endoscoped control patients. the specificity and sensitivity of the sero-diagnostic assay for histological gastritis in a ... | 1988 | 3166534 |
| prevalence of campylobacter pylori in duodenal and gastric mucosa--relationship to inflammation. | endoscopic biopsies from 310 patients were studied for the presence of campylobacter pylori (cp). in the first group of 150 patients duodenal and antral biopsies were obtained and in the second (160 patients) biopsies were taken from various sites in the stomach including the antrum, incisura, body and fundus. in the duodenum there was a strong association with active inflammation and the presence of gastric metaplasia. cp were never seen in the normal duodenum. in the stomach cp presence was as ... | 1988 | 3166535 |
| the efficacy of antimicrobial treatment in campylobacter pylori-associated gastritis and duodenal ulcer. | the efficacy of various antimicrobial and anti-ulcer agents on the elimination of campylobacter pylori in duodenal ulcer patients was investigated. ranitidine, cimetidine, pirenzepine, aluminium phosphate gel as well as combinations of h2-receptor antagonists or pirenzepine + penicillin v, ciprofloxacin, ofloxacin, phenyl-mercuryborate or rifampicin had no influence on c. pylori in vivo. short term elimination of c. pylori was achieved in 3/15 patients treated with ranitidine + bacampicillin and ... | 1988 | 3166537 |
| chronic erosive gastritis--a therapeutic approach with bismuth. | 37 patients with epigastric pain and chronic erosive gastritis underwent an open controlled therapeutic trial with bismuthsubsalicylate (bs). group a (21 patients) was treated with bs, liquid, 4 x 314 mg for three weeks, group b (16 patients) with bs tablets, 3 x 300 mg for two weeks. a significant reduction of symptoms (p less than 0.001) and endoscopically assessed chronic erosions (p less than 0.001) was achieved in both groups. campylobacter pylori was detected in 89% of the patients before ... | 1988 | 3166538 |
| epidemiology of campylobacter pylori infection: ethnic considerations. | a proper perspective of the association of campylobacter pylori with various diseases can be achieved only when the incidence of c. pylori infection in the normal population is known. we used the 13c urea breath test (a simple, specific, noninvasive, safe, and reproducible method to assess the presence of active c. pylori infection) to compare the frequency of c. pylori infection in healthy young adults from three areas of the world. we studied 26 women and 32 men (ages 20 to 29): 29 from the un ... | 1988 | 3166539 |
| pharmacokinetic, bacteriological and clinical aspects on the use of doxycycline in patients with active duodenal ulcer associated with campylobacter pylori. | seven patients with active duodenal ulcer and positive cultures for campylobacter pylori were given anti-ulcer treatment for 28 days with the addition of doxycycline for the last nine days. repeated endoscopy with biopsy for culture and doxycycline concentration determinations from the mucosa at the end of doxycycline therapy showed that five of seven patients remained positive in culture. the concentrations of doxycycline in biopsy material were 0.1 to 0.3 mg/kg in four patients; in three patie ... | 1988 | 3166544 |
| campylobacter pylori in upper gastrointestinal disease. | | 1988 | 3167290 |
| distribution of campylobacter pylori in dyspeptic and non-dyspeptic gastroenterologic patients. | the presence of campylobacter pylori (cp) in antral biopsies from patients undergoing routine gastroscopy or ercp was determined by microbiological and histological methods and correlated to the histopathology. 322 endoscopies were performed. of 125 cp positives only one did not present with histopathological evidence of gastritis. in patients with gastritis the percentage positive for cp was highest (88.6) in severe gastritis. we also found a highly significant correlation of cp to all types of ... | 1988 | 3168555 |
| [drug therapy of campylobacter pylori-associated gastritis]. | | 1988 | 3168772 |
| relationship between campylobacter pylori and gastritis in healthy humans after administration of placebo or indomethacin. | endoscopic and microscopic appearances of antral and fundic mucosa were correlated with the presence or absence of campylobacter pylori--and with plasma immunoglobulin g antibodies to that organism--in 23 healthy volunteers, 12 of whom had received indomethacin and 11 of whom had received no medication. antral c. pylori, found in 9 of 23 biopsy specimens (3 of 11 controls, 6 of 12 indomethacin-treated patients; not significantly different), correlated strongly with histologic evidence of active ... | 1988 | 3169487 |
| isolation of a spiral-shaped bacterium from the cat stomach. | a spiral- or helix-shaped bacterium that colonizes the stomachs of cats has been isolated in pure culture for the first time. the organism is tightly coiled with tufts of 10 to 17 polar flagella positioned slightly off center at the end of the cell. the body of the cell is entwined with unique periplasmic fibrils that usually occur in pairs, although groupings of one and three fibrils were also seen. the organism is strongly urease, catalase, and oxidase positive and is likely to belong to an as ... | 1988 | 3169989 |
| gastric colonization by campylobacter pylori subsp. mustelae in ferrets. | campylobacter pylori subsp. mustelae was cultured from both normal and inflamed gastric mucosa of ferrets. examination of neonatal, juvenile, and adult ferrets established that the gastric mucosa in the majority of preweanling (age, less than 6 weeks) ferrets sampled were not colonized with c. pylori subsp. mustelae, whereas the gastric mucosa of 100% of adult ferrets were colonized with this gastric organism. c. pylori subsp. mustelae was isolated from the gastric mucosa on a sequential basis f ... | 1988 | 3169994 |
| failure of ciprofloxacin to eradicate campylobacter pylori from the stomach. | | 1988 | 3170399 |
| the urease enzymes of campylobacter pylori and a related bacterium. | the urease enzyme of campylobacter pylori was studied and compared with that of a related spiral-shaped bacterium, st1, isolated from the rodent ileum. both bacteria possessed constitutive urease enzymes with activities up to 20-70 times that of proteus vulgaris. this activity was retained on sds-polyacrylamide gels. a major catalytic subunit of mol. wt 300,000 was located for all (six) strains of c. pylori subjected to sds-page whereas st1 had two active forms of mol. wts 140,000 and 150,000. w ... | 1988 | 3172169 |
| isolation of a gastric campylobacter-like organism from the stomach of four rhesus monkeys, and identification as campylobacter pylori. | campylobacter-like organisms, isolated from the gastric antrum of rhesus monkeys, were compared with campylobacter jejuni and c. pylori. they were similar to c. pylori by light microscopy, in ultrastructural morphology, in enzymic, fatty-acid-methyl-ester, and protein-profile analysis, and in antigenic reactivity with rabbit antisera to c. jejuni and c. pylori and with c. pylori-specific monoclonal antibody. because this natural infection of the rhesus monkey is associated with chronic gastritis ... | 1988 | 3172170 |
| immunoblot fingerprinting of campylobacter pylori. | one hundred and fifty isolates of campylobacter pylori were divided into nine groups by immunoblot fingerprinting. all isolates were typable and reproducibility between immunoblots was good provided both antigen and antibody preparation were standardised. discrimination was a problem as groups 1 and 2 accounted for 66% of isolates. recurrence after bismuth treatment was due to indistinguishable strains in five patients and different strains in three patients. | 1988 | 3172174 |
| [relation between chronic antral gastritis and the presence of campylobacter pyloridis. retrospective study on a geriatric population]. | | 1988 | 3173758 |
| [campylobacter pylori and gastric mucosa. what method to use in the diagnosis?]. | | 1988 | 3175273 |
| na+/h+ ion-exchange property of postmortem human gastric mucus. | the na+/h+ ion-exchange property of human gastric mucus and its relationship to age has been studied. mucus was collected from 40 postmortem human stomachs (age range, 21-83 years) within 24 h of traumatic death. twenty-one stomachs (age range, 21-76 years) were free of campylobacter pylori infection and histologic gastritis. mucus from these stomachs was consistently a cation exchanger at ph 6 in an in vitro system. the cation-exchange capacity of mucus from seven stomachs within the age range ... | 1988 | 3175531 |
| campylobacter pylori in peptic ulcer disease. ii. endoscopic findings and cultivation of c. pylori. | specimens from 153 consecutive patients were cultivated for c. pylori, and findings were correlated to the endoscopic findings. c. pylori was cultivated more frequently from males than from females. culture-positive males had a high frequency of prepyloric abnormalities. no correlation between age or ph in gastric juice and cultivation of c. pylori was found. c. pylori was found significantly more often in patients with peptic ulcer and/or duodenitis than in patients with normal endoscopic findi ... | 1988 | 3175537 |
| campylobacter pylori--associated gastritis and peptic ulcer disease in children. | specimens obtained at gastric biopsies performed for suspected acid peptic disease in patients 5 through 17 years of age were retrospectively reviewed for the presence of campylobacter pylori (cp), a gram-negative bacillus associated with chronic gastritis and peptic ulcer disease in adults. of 98 patients who underwent antral biopsy (the most reliably colonized site in the stomach), 40 had chronic gastritis histologically. of those 40 patients, 22 (55%) had cp present on the gastric surface. no ... | 1988 | 3177319 |
| [campylobacter pylori, ulcer and dyspepsia]. | | 1988 | 3181030 |
| is transmission of campylobacter pylori by endoscopic examination possible? | | 1988 | 3181090 |
| the characterization and pathological significance of gastric campylobacter-like organisms in the ferret: a model for chronic gastritis? | gastric campylobacter-like organisms (clo) were isolated from gastric tissues removed at sacrifice from 17 mature ferrets; all animals were colonized, but no macroscopic mucosal lesions or histological features of chronic gastritis were seen. the isolates resembled campylobacter pylori in many cultural and biochemical characteristics, and produced substantial urease activity, but there were sufficient differences from c. pylori to suggest that ferret gastric clo represents a separate species. co ... | 1988 | 3181311 |
| gastric acid secretion and campylobacter pylori. | | 1988 | 3181692 |
| effects of antibacterial treatment of campylobacter pylori-associated gastritis in children. | | 1988 | 3181694 |
| in vitro model for campylobacter pylori adherence properties. | the adherence of 12 strains of campylobacter pylori was studied on four cell lines. immunofluorescence and scanning and transmission electron microscopy were used to visualize the bacteria. a heavy adherence to the epithelial cell line hep-2 and to the intestinal cell line int-407 was noted. by transmission electron microscopy, a close association between bacteria and cells in the form of cup-like structures was observed, but pedestals were not present. | 1988 | 3182085 |
| campylobacter pylori: what is it doing there? | | 1988 | 3182639 |
| campylobacter pylori isolated from the stomach of the monkey, macaca nemestrina. | campylobacter pylori was isolated from the gastric mucosa in 6 of 24 pigtailed macaques (macaca nemestrina) examined by gastric biopsy and culture; 3 isolates were recovered during gastroendoscopy, and 3 were recovered at necropsy. the isolates were morphologically and biochemically similar to the human type strain nctc 11638, differing only in colony diameter, pigmentation, and rate of growth. identity of the isolates was confirmed by whole-genomic dna-dna hybridization with the type strain. co ... | 1988 | 3183019 |
| treatment of campylobacter pylori-associated antral gastritis in children with bismuth subsalicylate and ampicillin. | we evaluated the efficacy of medical therapy, consisting of liquid bismuth subsalicylate prescribed either in combination with oral ampicillin (n = 15) or alone (n = 1), in the clearing of bacterial colonization on the antrum in 16 children with campylobacter pylori-associated antral gastritis. we also examined the effects of medical treatment on altering the severity of associated antral inflammation. eight patients had upper gastrointestinal tract hemorrhage, two had acute gastric outlet obstr ... | 1988 | 3183851 |
| [campylobacter pylori colonization of duodenal and gastric mucosa in duodenitis and duodenal ulcer]. | | 1988 | 3185416 |
| [campylobacter pyloridis in chronic gastritis and peptic ulcer]. | | 1988 | 3187687 |
| [campylobacter pylori. a causative agent of gastritis and ulcer disease or a commonly associated agent?]. | | 1988 | 3188413 |
| campylobacter pylori infection. | | 1988 | 3190047 |
| incidence of campylobacter pylori in patients with upper gastro-intestinal symptoms. | antral mucosal biopsy specimens were examined microbiologically and histologically for the presence of campylobacter pylori in 224 patients with upper gastrointestinal symptoms. the gastric mucosa of 183 patients (82%) were found to harbour c. pylori. c. pylori was strongly associated with the presence of histological gastritis (93%) and was detected in only 10% of 30 patients in whom histological examination of gastric biopsy specimens was negative. endoscopically diagnosed duodenal lesions wer ... | 1988 | 3194804 |
| prevalence of gastric metaplasia, inflammation, and campylobacter pylori in the duodenum of members of a normal population. | mucosal biopsies were obtained from 116 asymptomatic volunteers (50% were male; mean age, 46 years; age range, 19-91 years) to study the prevalence of duodenal gastric metaplasia (gm) and its association with inflammation and campylobacter pylori in a normal population. gm was identified in 25 subjects (22%). eighty-three subjects (72%) had histologic duodenitis, but in only 10 did the infiltrate include neutrophils (grade 2 duodenitis). c. pylori was found in the stomach in 36 subjects (31%), a ... | 1988 | 3195501 |
| distribution of campylobacter pylori and gastritis in the stomach of patients with and without duodenal ulcer. | the presence of campylobacter pylori and histological changes of gastric mucosa were studied in 50 consecutive patients with gastric complaints. c. pylori was isolated from the antrum, body, and fundus of 40 patients with (n = 20) and without (n = 20) duodenal ulcer. the incidence of gastritis was not significantly different in the antrum of c. pylori-positive patients with and without ulcer. otherwise, oxyntic mucosa of both body and fundus regions exhibited gastritis in 64.1% of the c. pylori- ... | 1988 | 3195542 |
| campylobacter pylori in families of children with relapsing gastroduodenal disease due to c. pylori infection. | | 1988 | 3195559 |
| campylobacter pylori--gastroduodenal pathogen or opportunistic bystander? | | 1988 | 3196241 |
| campylobacter pylori, resistant duodenal ulcers, and antibiotic treatment. | | 1988 | 3199281 |
| campylobacter pylori and barrett's esophagus. | campylobacter pylori is thought to be confined to gastric mucosa; when detected in the duodenum in association with duodenal ulceration, the organism infects only areas of gastric metaplasia. barrett's esophagus is a metaplastic condition of the esophagus, in which areas or islands of "gastric-type" epithelium are found. to determine whether c. pylori colonized the esophagus of patients with barrett's esophagus, we studied retrospectively 23 unselected patients who had endoscopic and biopsy evid ... | 1988 | 3199885 |
| campylobacter pylori and barrett's esophagus. | occurrence of c. pylori infection of mucosa outside of the stomach might provide an ideal opportunity to examine c. pylori-mucosal interactions apart from the effects of acid and pepsin. techniques previously used to examine barrett's epithelium (for example, special mucin stains or scanning and transmission electron microscopy) might be particularly useful for exploration of new associations and formulation of new hypotheses. whether c. pylori has a role in development of barrett's ulcer or ade ... | 1988 | 3199889 |
| the use of serology to diagnose active campylobacter pylori infection. | a serological test that predicts accurately active campylobacter pylori infection in the human stomach has been developed and validated by means of serum from 189 patients who were undergoing endoscopy in sydney. our enzyme-linked immunosorbent assay (elisa) has a sensitivity of 100% and a specificity of 94%. an important part of the test is the inclusion of a simple absorption step with c. jejuni for those sera whose results are close to the cut-off point for positivity. this has been shown to ... | 1988 | 3200186 |
| [campylobacter pylori colonization and gastroduodenal pathology]. | 355 patients suffering from dyspepsia with an endoscopic pathological picture were examined. campylobacter pylori was sought histologically and by culture. histological and cultural results were compared. there were no bacteria on the gastric mucosa of 46 patients who had no histopathological evidence of lesions. cultural studies were performed in 40 cases on endoscopic instruments used during the examination, before and after standard sterilization. campylobacter pylori was found on the gastric ... | 1988 | 3200472 |
| susceptibility of campylobacter pylori to macrolides and fluoroquinolones. | the in-vitro activities of several 14-, 15- and 16-membered macrolides and fluoroquinolones against campylobacter pylori were determined. in general, 14-membered macrolides, such as clarithromycin and flurithromycin, were more active than the 15-membered macrolide, azithromycin, which was more active than 16-membered macrolides, such as miocamycin and rokitamycin. fluoroquinolones, except ciprofloxacin and a-61827, were less active than macrolides. clarithromycin was the most active of the new c ... | 1988 | 3209524 |
| inhibitory antimicrobial concentrations against campylobacter pylori in gastric mucosa. | gastric mucosal concentrations of erythromycin, amoxycillin, ampicillin and ciprofloxacin were determined in patients at upper gastrointestinal endoscopy 38-480 min after a 500 mg dose of erythromycin ethyl succinate, erythromycin stearate, amoxycillin, pivampicillin or ciprofloxacin. all the agents attained concentrations greater than the mic 90 for campylobacter pylori. the macrolides attained the lowest concentrations. there was no significant difference between concentrations attained with e ... | 1988 | 3209531 |
| [campylobacter pylori, chronic gastritis and ulcer disease]. | | 1988 | 3217644 |
| [campylobacter pylori in chronic gastritis and the therapeutic efficacy of colloidal bismuthate]. | | 1988 | 3219933 |
| [antibiotics in the treatment of intractable duodenal ulcer disease complicated by campylobacter pylori infection]. | | 1988 | 3219934 |
| [campylobacter pylori in asymptomatic volunteers]. | | 1988 | 3219935 |