| duodenal ulcer relapse after eradication of campylobacter pylori. | | 1989 | 2564914 |
| campylobacter pylori, non-ulcer dyspepsia, and race in malaysia. | | 1989 | 2565493 |
| campylobacter pylori and duodenal ulcers: the gastrin link. | the possibility that campylobacter pylori (cp) in the gastric antrum stimulates gastrin release in duodenal ulcer (du) disease was examined in 31 patients. the 25 patients with antral colonisation with cp had higher basal and meal-stimulated plasma gastrin concentrations, and higher peak acid output (pao), than did the 6 without cp in the autumn. | 1989 | 2566737 |
| diagnostic value of an immunoassay to detect anti campylobacter pylori antibodies in non-ulcer dyspepsia. | an enzyme-linked immunosorbent assay (elisa) for detection of igg antibodies against campylobacter pylori was used to examine sera from 70 patients with non-ulcer dyspepsia. 48 patients had c pylori associated gastritis according to culture or histology; mean optical density (od) of the elisa was significantly higher than that for the 22 patients with normal antral mucosa and absence of c pylori. positive and negative predictive values for campylobacter-associated gastritis were 100% above od 2. ... | 1989 | 2566745 |
| is susceptibility to campylobacter pylori infection genetically determined? | | 1989 | 2567462 |
| effect of a campylobacter pylori protein on acid secretion by parietal cells. | the uptake of [14c]-aminopyrine by rabbit gastric epithelial cells was used as an indirect assay for acid secretion from parietal cells. campylobacter pylori strains, isolated from the stomachs of 3 patients with chronic gastritis, duodenal ulcer, and near-normal mucosa, respectively, inhibited acid secretion as effectively as 10(-4) mol/l cimetidine. the inhibition occurred with whole organisms and with sonicates. preliminary characterisation suggested that the inhibition was due to a nondialys ... | 1989 | 2568521 |
| campylobacter pylori and acid secretion. | | 1989 | 2568533 |
| trimipramine and other antipsychotics inhibit campylobacter pylori in vitro. | a series of conventional anti-ulcer drugs, tricyclic antidepressants and neuroleptics (and some cns non-active isomers) were tested in vitro for possible inhibition of campylobacter pylori. these bacteria are claimed to play an etiological role in peptic ulcer disease, at least in gastritis b. while cimetidine, famotidine, ranitidine and pirenzepine were inactive, all the antipsychotic agents and their isomeric derivatives were active to various degrees with ic50 of 26-59 microm. of special inte ... | 1989 | 2568631 |
| colloidal bismuth in the treatment of duodenal ulceration: the benefit for the patient. | colloidal bismuth subcitrate (cbs) heals gastric and duodenal ulcers better than placebo and at rates similar to those observed with the commonly used h2-antagonists. indeed, healing of duodenal ulcers may be more rapid than with cimetidine during the first month of treatment. when treatment is withdrawn, however, relapse is slower after cbs than after the h2-antagonists. the mechanism is uncertain but may be related to the ability of cbs to suppress campylobacter pylori infection. the clinical ... | 1989 | 2568684 |
| gastric glycerolipid as a receptor for campylobacter pylori. | a species was detected in the lipid extract of human red blood cells and human and pig stomach tissue which was specifically recognised by isolates of campylobacter pylori. the levels of this lipid were higher in human stomach antrum than in fundus and in adult compared with infant tissue samples. chemical treatment of the purified substance suggests that it is a novel glycerolipid. | 1989 | 2569053 |
| [campylobacter pylori colonization of the antrum: effect of gastrin, somatostatin, pancreatic polypeptide and neurotensin]. | campylobacter pylori (c.p.) infection is often found in patients with antral gastritis and peptic ulcer disease. pathophysiological links are still unclear, and we therefore tested the hypothesis whether c.p. affects the gastrointestinal peptides and thus influences gastric acid secretion and protective factors. 94 patients were examined by upper gi endoscopy and blood analyzed for gastrin, somatostatin, pancreatic polypeptide and neurotensin. biopsies of antral mucosa were investigated for c.p. ... | 1989 | 2569233 |
| campylobacter pylori, gastrin, and duodenal ulcer. | | 1989 | 2569578 |
| omeprazole, campylobacter pylori, and duodenal ulcer. | | 1989 | 2569579 |
| lowered gastrin and gastric acidity after eradication of campylobacter pylori in duodenal ulcer. | | 1989 | 2570202 |
| time-resolved fluoroimmunoassay for campylobacter pylori antibodies. | | 1989 | 2570214 |
| campylobacter pylori and acid secretion. | | 1989 | 2570259 |
| detection of campylobacter pylori infection. | | 1989 | 2570274 |
| campylobacter pylori, gastrin, acid secretion, and duodenal ulcers. | | 1989 | 2570297 |
| prostaglandins and campylobacter pylori. | | 1989 | 2571035 |
| [distribution and prevalence of campylobacter pylori in the stomach]. | we investigated the distribution and prevalence of campylobacter pylori in the stomach and duodenum. in this study, 500 biopsy specimens were obtained from 245 patients. in each case, biopsy specimens were taken from more than 2 sites. c. pylori was detected by culture, urease test and acridine-orange stain. c. pylori was not detected on the intestinal metaplasia, gastric cancer tissue and duodenal mucosa without gastric metaplasia. in 21% of cases, c. pylori was detected in only one site. becau ... | 1989 | 2571739 |
| effect of various anti-ulcer drugs on rapid urease test for campylobacter pylori infection. | | 1989 | 2571835 |
| campylobacter pylori becomes helicobacter pylori. | | 1989 | 2572746 |
| the potential impact of campylobacter pylori on the treatment of duodenal ulcer disease. | in most studies the association between campylobacter pylori infection, type b gastritis and duodenal and gastric ulceration is extremely strong. the best evidence for it having an aetiological role is at present in type b gastritis. it should be remembered, however, that serological studies show carriage of the organism to be common also in the general population. an attempt is made here to gain some idea of the clinical impact of c. pylori infection in duodenal ulcer disease by analysing clini ... | 1989 | 2573145 |
| [campylobacter pylori infection in chronic gastritis and gastric cancer]. | the present paper analyses, in correlation with histologic lesions, the incidence of infection with campylobacter pylori (cp) in patients with chronic gastritis, operated stomach, gastric neoplasm and healthy subjects. in chronic gastritis, incidence of infection with cp is 71.6%, significantly increased in comparison with the controls, in whom the positive percentage is 37.5. the most of the patients in this group suffered from superficial chronic gastritis. in the group with operated stomach, ... | 1989 | 2573924 |
| evidence of gastritis with several helicobacter pylori strains. | | 1989 | 2574351 |
| omeprazole, helicobacter pylori, gastritis, and duodenal ulcer. | | 1989 | 2574352 |
| [helicobacter pylori-induced cost-benefit risk considerations]. | | 1989 | 2575693 |
| campylobacter pylori and gastroduodenal ulcer disease. a prospective study in a swedish population. | in a consecutive prospective series of 208 swedish primary peptic ulcer patients, 146 gastric, 55 duodenal and 7 in both sites, gastroduodenitis was found in 97.6% of the cases. the mucosal inflammation was associated with cp in 87% and 91% of the gastric and duodenal ulcer cases respectively. no significant correlation was found between cp colonisation and the type or severity of mucosal inflammation. gastric metaplasia was present in only 8% of 48 bulbar ulcer cases. ulcer healing and eradicat ... | 1989 | 2575789 |
| natural history of campylobacter pylori in duodenal ulceration treated with an h2-antagonist. | campylobacter pylori has been associated closely with active antral gastritis and duodenal ulcer but its pathogenetic role remains uncertain. the present longitudinal study examined, prospectively, the endoscopic antral biopsies of 45 patients with duodenal ulceration, taken before and after healing of the duodenal ulcer, during remission and at relapse. the biopsies were examined for the activity and degree of chronic inflammation of the gastritis and for the occurrence and density of c. pylori ... | 1989 | 2577695 |
| campylobacter pylori: prevalence and significance in patients with chronic renal failure. | since gastric symptoms are frequent in uremic patients, we have studied the prevalence and significance of campylobacter pylori infection in patients with chronic renal failure. the prevalence of serum igg antibodies to c. pylori in 50 patients on regular dialysis treatment (44%) was similar to that of a control group of 40 blood donors (45%), but was significantly lower (p = 0.0001) than that in 31 patients with peptic ulcer disease (93%). furthermore, 11 of the 50 patients with chronic renal f ... | 1989 | 2582650 |
| campylobacter pylori is not associated with gastroparesis. | there is a high incidence of campylobacter pylori in the gastric mucosa of patients with duodenal ulcer, gastric ulcer, and nonulcer dyspepsia. factors that lead to development of this infection are unknown. we hypothesized that delayed solid-phase gastric emptying, a condition characterized by antral stasis, might predispose to campylobacter pylori infection. we prospectively studied 51 patients with symptoms of gastroparesis using a solid-phase gastric emptying study and upper endoscopy. patie ... | 1989 | 2582980 |
| ultrastructure of another spiral organism associated with human gastritis. | campylobacter pylori may not be the only organism that causes active chronic gastritis in man. we report two cases of gastric infection with a spiral organism distinct from c. pylori. the first patient is a 36-year-old female who presented with epigastric pain and abdominal colic present since childhood and who had 14 cats. endoscopy was normal. the second patient kept two dogs. histology of gastric mucosal biopsy specimens in both patients revealed active chronic gastritis, most severe in body ... | 1989 | 2582989 |
| campylobacter pylori in esophagus. | | 1989 | 2582992 |
| [antibiotics for campylobacter pylori colonization of the stomach]. | | 1989 | 2583013 |
| profound spatial clustering of simultaneous peptic ulcers. | in an endoscopic study of 90 consecutive patients with more than one peptic ulcer, the ulcers in an individual were profoundly spatially clustered. clustering of ulcer locations was shown using a non-parametric test of clustering (kruskal-wallis statistic with 89 degrees of freedom = 151.31, probability of observing this extreme statistic with no clustering less than 0.0005) and a parametric test of clustering (f test statistic with 89 and 124 degrees of freedom = 5.41, probability of observing ... | 1989 | 2583560 |
| patterns of colonisation of campylobacter pylori in the oesophagus, stomach and duodenum. | thirty five subjects underwent upper gastrointestinal endoscopy and multiple biopsy (30 patients, five normal subjects). a total of 11 biopsies per subject from four sites (oesophagus (three), gastric body (two), antrum (three), duodenum (three] were examined for inflammation and the presence of campylobacter pylori and using standard methods of culture and by light (lm) and electron microscopy (em). the organism was cultured from oesophageal biopsies in eight of 30 (27%) patients but could not ... | 1989 | 2583561 |
| duodenal gastric heterotopia and campylobacter pylori: an exception to the rule? | | 1989 | 2584414 |
| prevalence of helicobacter pylori infection and histologic gastritis in asymptomatic persons. | we estimated the prevalences of helicobacter pylori (formerly called campylobacter pylori) infection and histologic gastritis in 113 asymptomatic persons, using endoscopic biopsy of the gastric antrum and corpus. unsuspected lesions, mainly mucosal erosions, were revealed at endoscopy in 16 subjects (14 percent). gastritis was found in 42 subjects (37 percent), of whom 36 (32 percent of the total) were found to be infected with h. pylori on the basis of hematoxylin-eosin staining. h. pylori was ... | 1989 | 2586553 |
| trends in campylobacter pylori in pediatric and adult antral biopsies. a 5-year retrospective analysis. | campylobacter pylori has been associated with chronic gastritis and antral ulceration in adults and has recently been reported in children with primary antral gastritis and duodenal ulceration. we reviewed all gastric antral biopsy specimens from children (n = 30) and adults (n = 77) over the past 5 years at the university of california, san francisco. tissue sections were stained with giemsa to detect c pylori, and medical histories were obtained by chart review. the prevalence of c pylori in a ... | 1989 | 2588580 |
| the source of campylobacter pylori. | sera from 98 abattoir workers were tested for igg as well as for iga to campylobacter pylori, c. jejuni and klebsiella. clerical workers had significantly lower c. pylori and c. jejuni antibody levels than any of the groups in direct contact with freshly cut animal parts. no difference was found for antibodies to klebsiella. twenty-nine non-clerical workers with high igg antibody levels against c. pylori consented to upper gastrointestinal endoscopy. c. pylori associated gastritis was found in a ... | 1989 | 2590720 |
| evidence for the occurrence of the same strain of campylobacter pylori in the stomach and dental plaque. | restriction endonuclease analysis with hindiii, haeiii, and bglii endonucleases of dna extracted from each of eight colonies of campylobacter pylori subcultured from a stomach biopsy and from each of eight colonies subcultured from dental plaque of the same patient indicated that at least three strains were present in the dental plaque but only one strain was present in the biopsy. one of the dental strains had restriction patterns indistinguishable from those of the biopsy isolate, providing ev ... | 1989 | 2592545 |
| cellular and humoral immune responses in campylobacter pylori-associated chronic gastritis. | gastric cellular and humoral immune response investigated by immunoperoxidase staining of 53 antral biopsies showed significant differences in campylobacter pylori-associated gastritis as compared with non-bacterial chronic gastritis and normal controls. iga, secretory component, and complement c3 coated bacilli were seen in all cases of active chronic gastritis. c3 was always associated with coating by iga, igm, or both, which were rarely seen in gastritis without polymorphonuclear neutrophil i ... | 1989 | 2593047 |
| gastroduodenal pathology and campylobacter pylori. second meeting of the european campylobacter pylori study group. october 12-14, 1989, ulm, frg. abstracts. | | 1989 | 2593601 |
| campylobacter pylori infection: diagnosis and therapy. | | 1989 | 2593955 |
| the 14c-urea breath-test for the detection of gastric campylobacter pylori infection. | a breath-test has been developed for the detection of gastric infection with campylobacter pylori. urea that is labelled with carbon 14 is administered to a fasting patient and the patient's breath is sampled for radioactivity over the following 30 minutes. if c. pylori is present in the patient's stomach, urease activity causes hydrolysis of the urea and the 14c is absorbed as carbon dioxide. this carbon dioxide enters the patient's bicarbonate pool and eventually is excreted in the breath. the ... | 1989 | 2593958 |
| treatment of campylobacter pylori does not alter gastric acid secretion. | the effect of treatment of campylobacter pylori-associated gastritis on acid secretion was studied to examine further the strong association between c. pylori and peptic diseases. twelve symptomatic patients with non-ulcer dyspepsia and c. pylori-associated histologic gastritis had basal and pentagastrin-stimulated gastric acid analysis before and after a 14-day course of amoxicillin 250 mg qid and bismuth subsalicylate 524 mg qid. endoscopy, antral biopsies, and symptom questionnaires were obta ... | 1989 | 2596452 |
| prospective comparative study of the influence of postoperative bile reflux on gastric mucosal histology and campylobacter pylori infection. | biopsies of 17 peptic ulcer patients, randomly treated by partial gastrectomy with either billroth-ii (n = 9) or roux-en-y (n = 8) anastomosis were studied before and six months after surgery to determine the role of bile reflux in the early postoperative histological alterations of the gastric mucosa. after bii-gastrectomy bile acid reflux (median 16.1 mumol/h) was significantly higher (p less than 0.0001) than after ry-gastrectomy (0.1 mumol/h). campylobacter pylori was present in the preopera ... | 1989 | 2599441 |
| [campylobacter pylori and diseases of the stomach]. | in spite of extensive clinical studies the pathogenicity of campylobacter pylori in various diseases of the stomach is still not fully elucidated. in peptic ulcer campylobacter pylori has not provided a reasonable explanation for the localized and solitary development of ulcer lesion which is one of characteristic features of this clinical entity. our previous clinical study revealed that relapse occurs mostly at or near the scar of the preceding ulcer lesion and occurs more often in red scar th ... | 1989 | 2601081 |
| campylobacter pylori in dyspeptic subjects: a report from north india. | | 1989 | 2603193 |
| [bismuth--an environmental pollutant of the future]. | campylobacter pylori had become an interesting object in the discussion of the etiology of gastritis and peptic ulcer. marshall was the first to describe antrum gastritis and duodenal ulcers as infectious diseases. consequently new forms of therapy like antibiotics and bismuth salts were studied. in time the most favoured method of treatment is the administration of inorganic bismuth salts. there is a lack of recognition about the behaviour of this heavy metal in environment. 0.5 to 1 gram pure ... | 1989 | 2604843 |
| 'corkscrew-like' bacteria associated with gastritis. | gastric spiral bacteria morphologically unlike campylobacter pylori have recently been described in patients with gastritis. we report an additional case. these organisms are as yet unnamed, remain uncultured and are morphologically similar to organisms present in the gastric mucosa of various mammalian species. their possible role in the pathogenesis of gastritis is discussed. | 1989 | 2606459 |
| campylobacter-like organisms, nonsteroidal anti-inflammatory drugs and gastric lesions in patients with rheumatoid arthritis. | a histological study was performed in order to evaluate the prevalence of campylobacter-like organisms (clo) and gastric antral lesions in 85 rheumatoid arthritis (ra) patients using nsaids, and in 100 nonrheumatoid outpatients comparable in terms of sex and age, not using nsaids. histological evidence of gastritis was a common finding both in ra patients (88.2%) and in nonrheumatoid outpatients (89.0%). on the other hand, clo were detected in a significantly lower proportion (p less than 0.001) ... | 1989 | 2612748 |
| isotype evolution in the follow-up study of patients with campylobacter pylori associated gastritis. | four sequential immuno-assays were performed from may to november 1988 to follow the levels of igg, iga and igm to campylobacter pylori in 16 infected patients with histologically proven gastritis, among which 12 received appropriate therapy. histopathological examination of antral biopsies, bacteriological cultures and urease tests were performed on each occasion when serum was tested for antibodies to c. pylori. the detection and quantitative assessment of the various isotypes to this bacteriu ... | 1989 | 2612831 |
| interaction of campylobacter pylori... | | 1989 | 2612992 |
| campylobacter pylori in saudi patients undergoing upper gastrointestinal endoscopy: prevalence and effect of conventional therapy. | patients with symptoms of gastritis or peptic ulcer disease were recruited to study the prevalence of campylobacter pylori. on the basis of the endoscopic diagnosis only, the isolation rates of the organisms in normal, gastritis or gastroduodenitis (gd), and peptic ulcer (pu) disease patients, were not significantly different among the 89 patients evaluated. however, analysis based on histopathological evaluation (in 73 out of these 89 patients) revealed a significant difference (p = 0.00016) in ... | 1989 | 2613174 |
| rapid diagnostic test of campylobacter pylori infection in patients of gastritis, duodenal ulcer and gastric ulcer. a study of 100 cases. | campylobacter pylori has been cultured from 85-90% of antral biopsies of patients of gastritis, duodenal ulcer and gastric ulcer at different centres in the world. it has been now firmly implicated in the aetiology of active gastritis as well as suspected to cause repeated recurrences of peptic ulcers. however, the organism is very fastidious and is difficult to grow by standard culture methods as a result of which low positivity is often obtained even in well equipped centres. the rapid biopsy ... | 1989 | 2613635 |
| lymphoid follicles in antral mucosa: immune response to campylobacter pylori? | the prevalence of lymphoid follicles in endoscopic biopsy specimens from normal antral mucosa (n = 220), mucosa with reflux gastritis (n = 104), and in cases with campylobacter pylori-associated gastritis (n = 2544) was studied. in the latter group whether there were associations between degree and activity of gastritis and the prevalence of lymphoid follicles and between the occurrence of lymphoid follicles and the presence of intestinal metaplasia in the antrum were investigated. in cases with ... | 1989 | 2613920 |
| investigation of the relationship between gastric antral inflammation and campylobacter pylori using graphic tablet planimetry. | gastric antral endoscopic pinch biopsies from a group of dyspeptic patients were analysed for acute and chronic inflammatory cell numbers in the lamina propria and surface epithelial layer using computer-linked graphic tablet planimetry, and independently graded for campylobacter pylori (cp) infection using a visual scoring system with grade 1 assessed as patchy epithelial infection and grade 2 as a continuous layer of organisms on the mucosal surface extending into gastric pits. the study group ... | 1989 | 2614572 |
| [the role of campylobacter pylori in the clinical course of chronic antral gastritis and duodenal ulcer]. | | 1989 | 2615309 |
| [campylobacter pylori in endoscopic biopsies of the gastric antrum and its association with chronic gastritis]. | a prospective study of 156 patients with upper gastrointestinal complaints submitted to endoscopy with biopsy of the gastric antral mucosa, was performed in order to evaluate by light microscopy the presence of chronic gastritis and campylobacter pylori and its relationship to peptic ulcer. thirty were histologically normal, and 126 showed evidence of gastritis. ninety-four (74.6%) of the specimens with evidence of gastritis contained spiral bacteria whereas only three (10%) of the thirty gastri ... | 1989 | 2616983 |
| rapid diagnosis of campylobacter pylori by brush cytology. | campylobacter pylori (cp) was demonstrated in 67.2% of 67 patients using cytology as compared to 65.7% by histology. brush cytology is thus found to be superior to all other methods available to date for the detection of cp. it is rapid, simple, and has a high specificity. | 1989 | 2617160 |
| campylobacter pylori and non-ulcer dyspepsia. 2. a prospective study in a swedish population. | in a consecutive prospective series of 186 swedish persons with the diagnosis of non-ulcer dyspepsia 71.5% were found to have gastritis and/or bulbar duodenitis in endoscopic biopsies. gastroduodenitis was associated with campylobacter pylori (cp) in 83.5% of the cases. the double therapeutic approach using an antibiotic and a preparation containing bismuth in an uncontrolled therapeutic pilot trial resulted in improvement of the histological picture, disappearance of cp and amelioration of symp ... | 1989 | 2617168 |
| does omeprazole improve antimicrobial therapy directed towards gastric campylobacter pylori in patients with antral gastritis? a pilot study. | this double-blind pilot study has been undertaken in order to investigate the effect of amoxicillin and pronounced suppression of gastric acid secretion on mucosal colonisation with campylobacter pylori (cp). twenty four cp-positive patients were included in the study and assigned to 14 days of treatment in either one of the following three therapy groups: group 1: omeprazole 40 mg o.m. + amoxicillin 750 mg b.i.d (9 pat); group 2: omeprazole 40 mg o.m. (8 pat); group 3: amoxicillin 750 mg b.i.d ... | 1989 | 2617169 |
| campylobacter pylori in gastric, duodenal and jejunal juices and mucosae of patients with duodenal ulcer. | the presence of campylobacter pylori was investigated in biopsies and fluids obtained in the antrum, duodenal bulb and jejunum during jejunoscopy in 20 patients with an active duodenal ulcer. c. pylori was present in cultured antral biopsies in all patients, in the bulb of most patients (16/20), but was unusual in jejunal mucosa (2/20). using a non-contaminated sampling method of fluid, c. pylori was found in only two samples at each level. in conclusion, c. pylori is frequent in bulbar mucosa o ... | 1989 | 2618533 |
| [erosive duodenitis. clinical study of 287 cases]. | we relate here the results of a retrospective (1984-1987) investigation on 1043 protocols of gastroscopy, where the diagnosis of erosive duodenitis (ed) has been made 372 times in 287 patients. the presence of campylobacter pylori was not investigated. ed endoscopic incidence was 5.6%. patients were divided into three groups: 1. ed alone: 15.7%. 2. ed associated with duodenal ulcer (du): 48.4%. 3. e.d. associated with various diseases: 35.9%. there was a higher male/female ratio in the three gro ... | 1989 | 2618534 |
| campylobacter pylori colonization factor shows specificity for lactosylceramide sulfate and gm3 ganglioside. | the specificity of campylobacter pylori cell surface lectin, a presumptive colonization factor, was investigated using various sulfated and sialic acid containing glycolipids. c. pylori cells, cultured from human antral mucosal biopsies, were incubated with intact and modified glycolipid preparations and examined for agglutination inhibition of human erythrocytes. titration data revealed that the inhibitory activity was highest with lactosylceramide sulfate and gm3 ganglioside, while galactosylc ... | 1989 | 2619758 |
| [campylobacter pyloridis (cp) infection of gastric mucosa in the high and low risk areas of gastric cancer in liaoning province]. | gastric biopsies in 690 subjects from the high and low risk areas of gastric cancer were examined for identification of cp in the gastric mucosa by warthin-starry, gimenez and gram's stains. the result showed that the positive rate was 60-62% in the high risk area whereas it was only 12.6% in the low risk area in liaoning province. 81-85% of the positive subjects had active chronic gastritis including chronic superficial and atrophic gastritis. the result indicates a close correlation between th ... | 1989 | 2620634 |
| [campylobacter pylori associated gastritis]. | | 1989 | 2621061 |
| activity of quinolones against campylobacter pylori in vitro. | | 1989 | 2621174 |
| [morphology of chronic gastritis associated with campylobacter pylori infection]. | recently, a close relation has been found between infection of the gastric mucosa by campylobacter pylori and chronic gastritis. to establish the possible existence of characteristic morphologic changes in this disease, which can be differentiated from other unrelated forms of gastritis, we analyzed the antral biopsies obtained from 75 patients, 35 with duodenal peptic ulcer and 40 with nonulcerous dyspepsia. the diagnosis of c. pylori infection is based on positive biopsy culture or, if not, wh ... | 1989 | 2623310 |
| [reflections on the significance of detecting campylobacter pylori in a biopsy of the stomach mucosa]. | the inevitable confrontation with campylobacter pyloridis implicates the surgeon to take into consideration this germ. for that reason we have examinated campylobacter-positive patients with regard to histological changes of the gastric mucosa and to clinical symptoms. apart from the fact, that there was no campylobacter colonisation in the healthy gastric mucosa, there were not found specific features for the germ. | 1989 | 2624004 |
| [zhongxu-qizhi type of chronic gastritis and campylobacter pyloridis infection]. | according to the syndrome differentiation of tcm, one hundred cases of chronic gastritis were recognized as zhongxu-qizhi type (group i, 57 cases) and the other types (group ii, 43 cases), the latter further divided into 36 cases of disharmony of liver and stomach type and 7 cases of deficiency of stomach yin type. the pathohistological investigation and urease test showed that the campylobacter pyloridis (cp) infected rate in the group i (92.9%) was very significantly higher than that in group ... | 1989 | 2624982 |
| [effect of gastritis-granule on 52 cases of chronic gastritis infected with campylobacter pyloridis]. | 52 patients with chronic gastritis infected by campylobacter pyloridis (cp) were selected for random observation. the course of treatment was 4 weeks. in the group treated with granule-gastritis, the effective rate for cp was 81.3% (26/32), and the improvement rates of the symptoms and histopathological lesion were 87.5% (28/32) and 40.6% (13/32) respectively. in the group treated with wei-mei-su as the control group, the figures were 25%, 40% and 10% respectively. these differences were statist ... | 1989 | 2624985 |
| antral campylobacter pylori colonization, abo blood group, and secretor status. | | 1989 | 2625196 |
| [detection frequency of campylobacter pylori at the white exudate of gastric ulcers]. | | 1989 | 2625727 |
| value of serology (elisa and immunoblotting) for the diagnosis of campylobacter pylori infection. | fifty-two unselected patients referred to for upper gastrointestinal endoscopy were evaluated in several ways to determine the presence of campylobacter pylori. antibodies against this microorganism were measured to assess the value of serology for the diagnosis of c. pylori infection. five antral biopsy specimens were taken in each patient for culture and bacteriological determinations, histology [morphology and warthin-starry (ws) staining] and the urease test (2, 3 and 24 h). serum antibodies ... | 1989 | 2628136 |
| [campylobacter pylori and early relapse of peptic ulcer]. | a study is presented of campylobacter infestation of the gastric mucosa and frequency of recurrence of duodenal ulcer in these patients. it was established that maintained infestation of the gastric mucosa is despite healing of the ulcer an unfavourable prognostic factor concerning immediate recurrences of duodenal ulcers. early recurrences of the ulcer develop more frequently in patients with maintained infestation of the gastric mucosa with campylobacter pylori. | 1989 | 2629278 |
| [ulcer--campylobacter pylori--bismuth therapy]. | | 1989 | 2635701 |
| morphological evidence of campylobacter pylori pathogenicity in chronic gastritis and peptic ulcer. | campylobacter pylori (cp) were found in 84% of 384 patients with chronic gastritis and in none of 49 subjects without inflammation. cp were present in similar percentages among patients with active (90%) or healed peptic ulcer (84%), as well as in non-ulcerous dyspepsia complicated by gastritis (91%). cytoplasmic vacuolization and swelling of foveolar-superficial cells with adhering bacteria, micropapillae and microerosions were commonly found in cp-infected mucosa. in 100 cases with gastritis b ... | 1989 | 2637577 |
| simultaneous streptococcus pneumoniae, giardia lamblia and campylobacter pylori infection: an adult presentation of x-linked hypogammaglobulinaemia. | primary x-linked (bruton's) hypogammaglobulinaemia is uncommon. it usually presents clinically within the first two years of life--typically after the age of three months when maternal igg is exhausted. its diagnosis in middle life is exceptional. the presentation of the condition as a triple infection in a middle-aged man therefore seemed worthwhile reporting, as effective and safe prophylactic immunotherapy is now available. | 1989 | 2638574 |
| campylobacter pylori and peptic ulcer disease. | campylobacter pylori is now known to be the most common and important cause of gastritis, and c. pylori infections have been associated with duodenal ulcer, gastric ulcer, nonulcer dyspepsia, and gastric cancer. although it has been only possible to culture c. pylori for about 5 yr, there are already sufficient data available to allow us to develop the basic framework that relates c. pylori gastritis to the causation of peptic ulcer disease. we review the data and propose mechanisms that implica ... | 1989 | 2642447 |
| initial therapy and relapse of duodenal ulcer: possible acid secretory mechanisms. | review of the four studies investigating the effect of initial therapy on ulcer relapse indicates that there is an increased rate of relapse after treatment with h2-receptor antagonists compared with placebo or other classes of drugs when combined. when individual drugs are considered, this appears to hold true for colloidal bismuth alone. there are several possible reasons for these differences. the most likely candidate mechanism to account for these differences is an alteration in gastric sec ... | 1989 | 2642449 |
| monoclonal antibodies for specific immunoperoxidase detection of campylobacter pylori. | monoclonal antibodies were raised against campylobacter pylori to detect the organism in clinical specimens by the immunoperoxidase technique. twenty-one hybridoma cultures obtained were screened on tissue sections of gastric mucosa to identify antibodies that did not cross-react with host cells. to select antibodies with high grade of specificity to c. pylori, hybridomas were also screened by an enzyme-linked immunosorbent assay against a wide panel of gram-negative bacteria. fifteen antibodies ... | 1989 | 2642876 |
| media for the culture of campylobacter pylori. | | 1989 | 2643300 |
| molecular cloning and expression of campylobacter pylori species-specific antigens in escherichia coli k-12. | a gene bank of campylobacter pylori dna in escherichia coli was constructed by cloning sau3a-cleaved dna fragments into the bacteriophage vector lambda embl3. the expression of c. pylori antigens was determined by screening the gene library with adsorbed c. pylori whole-cell rabbit antisera. one recombinant clone which reacted positively (lambda cp2) was studied further. immunoblot analysis with lambda cp2 showed a polypeptide band of 66 kilodaltons (kda) reacting antigenically with the adsorbed ... | 1989 | 2643578 |
| indirect immunofluorescence test and enzyme-linked immunosorbent assay for detection of campylobacter pylori. | an indirect immunofluorescence test (iif) has been developed for detecting campylobacter pylori in gastroduodenal biopsies. this test was compared with standard methods of c. pylori diagnosis, namely gram staining and urease test, in a study population of 226 patients; 121 of the biopsy specimens were cultured for c. pylori as well. c. pylori colonization was detected in 154 of 226 patients (68%) by at least one of these methods (iif, 96%; gram staining, 78%; urease test, 60%; cultivation, 55%). ... | 1989 | 2644295 |
| association of campylobacter pylori with induced expression of class ii transplantation antigens on gastric epithelial cells. | campylobacter pylori was identified with immunoperoxidase staining and a mouse monoclonal antibody directed against c. pylori in gastric biopsy specimens from 24 patients with gastritis. c. pylori was not found in gastric biopsy specimens from six subjects with histologically normal mucosa. the monoclonal antibody, which was reactive with a surface protein of approximately 20 kilodaltons, was found to be specific for c. pylori, and the immunoperoxidase staining proved to be more sensitive and ra ... | 1989 | 2645211 |
| [is campylobacter pylori responsible for ulcer disease?]. | | 1989 | 2647566 |
| [campylobacter pylori: diagnostic value of the urease test during endoscopy]. | the aims of this prospective study were a) to evaluate the diagnostic value of the urease test for the detection of c. pylori in gastric biopsy specimens, b) to specify the prevalence of c. pylori in a sample of 74 patients from the grenoble area undergoing upper gastrointestinal endoscopy, c) to analyze the density of bacteria according to the biopsy site (antrum, body, edges of ulcer), d) to demonstrate any possible correlation between the histologic state of the antral and body mucosa and the ... | 1989 | 2647570 |
| campylobacter pylori: a new pathogen. | | 1989 | 2647950 |
| age-dependent increase of campylobacter pylori antibodies in blood donors. | antibodies against campylobacter pylori were determined in 500 blood donors aged 18 to 65 years. acid extract from a c. pylori strain was used as antigen in enzyme immunoassay. the proportion of donors with high antibody titers increased with age. for igg antibodies it was 10% in the age group from 18 to 25 years but 60% in the group from 56 to 65 years; the increase for iga and igm antibodies was from 5 to 42% and from 7 to 21%, respectively. the geometric mean titers of those with high values ... | 1989 | 2648556 |
| campylobacter pylori: associations with antral and fundic mucosal histology and diagnosis by serology in patients with upper gastrointestinal symptoms. | we obtained a sample of serum and mucosal biopsies from the antrum and usually from the corpus of the stomach from 35 symptomatic patients during routine endoscopy to analyze for the relationship between campylobacter pylori infection, inflammation, and the diagnostic utility of a c. pylori igg antibody assay. c. pylori was identified prospectively by culture and/or silver stain in gastric biopsies from 24 patients, and the antibody was detected in 19 (79%) of these patients. the antibody assay ... | 1989 | 2648815 |
| suggestions for the rapid diagnosis of campylobacter pylori infection in endoscopic settings. | | 1989 | 2651099 |
| [antimicrobial drugs in the treatment of stomach and duodenal diseases associated with campylobacter pylori]. | | 1989 | 2654463 |
| [rapid detection and easy culture of campylobacter pylori from human gastric mucosa]. | 96 gastric mucosal biopsy specimens collected at endoscopy and placed into 3 ml 20% glucose were examined histologically, microscopically (gram staining), by urease test, and by cultivation. the rate of positive findings ws 62.5% by urease test, 34.4% by cultivation, and 27.0% microscopically. by using simple selective solid media, the number of positive cultivations increased to 58.7%. positive microbiological findings were established by one of three microbiological methods at a high percentag ... | 1989 | 2655826 |
| sucralfate and other non-antisecretory agents in the treatment of peptic ulcer disease. | sucralfate as well as colloidal bismuth subcitrate (cbs) and probably also bismuth subsalicylate (bbs) are effective in the acute treatment of peptic ulcer disease. sucralfate also has positive effects upon symptoms and healing of peptic lesions in reflux esophagitis. healing rates in gastric and duodenal ulcers are equal to those obtained with h2-antagonists. side effects are rare, transient and generally mild. therapy with bismuth compounds should be restricted to 4-8 weeks (cave: bismuth ence ... | 1989 | 2657280 |
| role of campylobacter pylori in the etiology of peptic ulcer. | campylobacter pylori has been associated with chronic gastritis type b and gastric and duodenal peptic ulcer. among the numerous works published in recent years, we intend to briefly review the principal characteristics of this association: microbiological characteristics of c. pylori and pathogenic aspects that can be implicated in the pathophysiology of the entities mentioned, i.e., biochemical factors, adherence, invasiveness, immunology, toxins. | 1989 | 2657283 |
| [is campylobacter pylori the pathogen of ulcer disease? pro]. | | 1989 | 2658389 |
| [is campylobacter pylori the pathogen of ulcer disease? contra]. | | 1989 | 2658390 |
| effect of sucralfate and cimetidine on duodenal ulcer-associated antral gastritis and campylobacter pylori. | the course of gastritis and campylobacter pylori was studied in a single-blind randomized trial comparing cimetidine 200 mg three times a day and 400 mg at night and sucralfate 1 g four times a day orally for four weeks in 140 patients with proved duodenal ulcer. at least two antral biopsies were performed during endoscopy before entry and at the end of four weeks. the activity and the degree of chronic inflammation, as assessed histologically by the degree of infiltration of, respectively, poly ... | 1989 | 2660558 |