Publications
| Title | Abstract | Year(sorted ascending) Filter | PMID Filter |
|---|
| [isolation of campylobacter pylori from antropyloric biopsies. a year of systematic research]. | 1988 | 2965362 | |
| [initial experience with the detection of campylobacter pylori]. | 1988 | 2967116 | |
| [campylobacter pyloridis and gastroduodenal inflammation]. | 1988 | 2972404 | |
| [do nonsteroidal anti-inflammatory drugs have a protective effect against campylobacter pylori?]. | 1988 | 2978311 | |
| campylobacter pylori-associated gastritis: attempts to eradicate the bacteria by various antibiotics and anti-ulcer regimens. | the efficacy of various antimicrobial and anti-ulcer agents on the eradication of campylobacter pylori in patients with antral gastritis or duodenal ulcers was investigated by several open studies or double-blind, placebo-controlled protocols. among the anti-ulcer agents, ranitidine, cimetidine or sucraflate had no effect on c. pylori. colloidal bismuth subcitrate achieved clearance of c. pylori in 40% of treated patients at the end of therapy but a high relapse rate (14/16 patients) was observe ... | 1988 | 2979039 |
| campylobacter pylori in swedish patients referred for gastroscopy. | campylobacter pylori was isolated more often from patient with peptic ulcers and in an age-related manner in a material of 395 consecutive patients referred for gastroscopy. direct microscopy was done with gram and acridine orange stain and found too insensitive for practical use. all patients were investigated serologically with an enzyme immunoassay which showed excellent correlation with positive cultures for c. pylori. the findings were discussed, and the enzyme immunoassay, with a negative ... | 1988 | 2449900 |
| improved visualisation of mucus penetration by campylobacter pylori using a brown-hopps stain. | 1988 | 2450896 | |
| campylobacter pylori--acridine orange stain and ultraviolet fluorescence. | 1988 | 2453440 | |
| campylobacter pylori detected by indirect immunohistochemical technique. | an immunohistochemical assay for staining c. pylori is described. the method is compared with cultivation of c. pylori and observation of campylobacter-like organisms (clos) in hematoxyline-eosine (he) stained sections. eighteen biopsies from which c. pylori was cultivated but not seen in he stained sections and three culture negative biopsies with clos seen in he stained sections were selected from 331 biopsies including 113 culture positive biopsies. there were agreements between cultivation o ... | 1988 | 2456087 |
| culture of the organisms and histochemical identification. | the detection of campylobacter pylori in the upper gastrointestinal tract has important implications for the pathogenesis and treatment of peptic disease. c. pylori is best isolated from biopsies using blood agar or chocolate blood agar incorporating a selective supplement. plates should be incubated under microaerophilic conditions for five days. a gram's stain of a biopsy smear provides a more rapid result but the organism may be patchy in its distribution. of the histochemical methods used, t ... | 1988 | 2458624 |
| n-acetylneuraminyllactose-binding fibrillar hemagglutinin of campylobacter pylori: a putative colonization factor antigen. | campylobacter pylori is the causative agent of gastritis and possibly of peptic and duodenal ulcers in adults. histological observations show c. pylori attached to gastric epithelium as well as in the mucus layer of the stomach. we found that clinical isolates of c. pylori possess a cell-bound hemagglutinin detectable with human erythrocytes (all phenotypes tested) and those of a variety of animal species. the c. pylori hemagglutinin is antigenic, heat sensitive, and destroyed by pronase and pap ... | 1988 | 2459065 |
| [effect of bismuth subsalicylate versus cimetidine on campylobacter pylori, ulcer healing and rate of recurrence]. | 1988 | 3043160 | |
| [campylobacter pyloridis and diseases of the stomach and duodenum]. | 1988 | 3046137 | |
| monoclonal antibodies for detection of campylobacter pylori in biopsy smears and frozen sections. | 1988 | 3047850 | |
| heterogeneity of campylobacter pylori as demonstrated by co-agglutination testing with rabbit antibodies. | the indirect immunofluorescence (ifl) and the co-agglutination (coa) methods were used to study the serology of campylobacter pylori strains isolated from patients in different countries (sweden, finland, canada and australia). antisera were obtained from rabbits immunized with whole cell antigens. with ifl tests the highest serum titers were obtained with c. pylori strains and their homologous antisera. these tests also showed that all the tested strains contained cross-reactive antigens. with ... | 1988 | 3047851 |
| comparison of different tests for campylobacter pylori. | 1988 | 3047852 | |
| bismuth: effects on gastritis and peptic ulcer. | the healing properties of colloidal bismuth subcitrate (cbs) on peptic ulcer are well established and several studies have shown that healing with cbs is associated with a lower relapse rate than that produced by h2-receptor antagonists. the recent observation that cbs is effective against campylobacter pylori has shed light on this because recent studies have shown that eradication of c. pylori by cbs leads to resolution of the associated gastritis and this may explain the low relapse rates. cb ... | 1988 | 3047853 |
| the role of antibiotics in campylobacter pylori associated peptic ulcer disease. | since the recognition and first culture of campylobacter pylori (cp) the hypothesis of a pathogenic role has been strengthened by numerous investigations. its close association with active chronic gastritis and even active duodenitis, and the disappearance of these pathologic conditions with antimicrobial treatment suggest a pathogenic role for cp in active chronic gastritis. the close association of antral gastritis with duodenal ulcer (du) suggests that cp associated active chronic gastritis m ... | 1988 | 3047854 |
| gastroduodenal complications of chronic nsaid therapy. | the fact that nonsteroidal anti-inflammatory drugs (nsaids) damage the gastroduodenal mucosa is no longer contested. endoscopic studies in normal volunteers after nsaid administration have failed to predict which nsaids would be safest when administered chronically. nsaid use has been associated with a disproportionately high frequency of upper gastrointestinal bleeding and perforation of ulcers. all of the newer nsaids appear to be similar in their propensity to cause mucosal damage, including ... | 1988 | 3048080 |
| bacteria in ulcer pathogenesis. | a great deal of information about the spiral bacteria of the stomach has accumulated in the past 5 years. these bacteria, currently named campylobacter pylori but likely to be renamed as a new genus, have adapted to living beneath the mucus layer and above the gastric surface mucous cells. when metaplastic gastric mucous cells are also present in the duodenal bulb, c. pylori may also get a foothold in this latter location. observations of the high prevalence of c. pylori in patients with gastrit ... | 1988 | 3048451 |
| a review of campylobacter pylori in upper gastrointestinal disease. | the organism campylobacter pylori is frequently found in association with gastritis and peptic ulcer disease. whether it is the cause, a contributory factor or a simple commensal is not known but there is evidence to support a pathological role. current research may alter our understanding of the causes of upper gastrointestinal disease and have important implications for treatment. | 1988 | 3048506 |
| nitrofurans in the treatment of gastritis associated with campylobacter pylori. the gastrointestinal physiology working group of cayetano heredia and the johns hopkins universities. | we conducted a double-blind, placebo-controlled, randomized treatment study in peruvian adults with antral gastritis associated with campylobacter pylori. patients received either 400 mg of furazolidone (n = 14), 400 mg of nitrofurantoin (n = 24), or a placebo (n = 31) for 14 days. endoscopy was carried out at baseline, 1 day after ceasing therapy, and 6 wk after the end of treatment to verify colonization by c. pylori and determine the extent of gastric inflammation. treatment with nitrofuranto ... | 1988 | 3049213 |
| opsonophagocytosis of campylobacter pylori. | the opsonic activity of human serum from various sources against campylobacter pylori was compared. all sera, whether from control subjects with no symptoms of gastritis or peptic ulceration, or from symptomatic patients from whom c. pylori had or had not been isolated, opsonised c. pylori equally well. opsonisation depended on the alternative pathway of complement activation but not on antibody. these findings suggest that antibody plays no role in protection against c. pylori and that the pres ... | 1988 | 3050112 |
| colloidal bismuth subcitrate. a review of its pharmacodynamic and pharmacokinetic properties, and its therapeutic use in peptic ulcer disease. | colloidal bismuth subcitrate (cbs) possesses at least equal efficacy with histamine h2-receptor antagonist drugs in the treatment of peptic ulcer disease. however, cbs has the advantage of slower ulcer relapse rates than those seen after initial healing with the h2-antagonists. it has been postulated that this effect may be partly due to the antibacterial properties of cbs against campylobacter pylori, a bacterium found in the gastric mucosa and gastric metaplasia within the duodenum of most pat ... | 1988 | 3053124 |
| campylobacter pyloridis is not responsible for duodenal ulcer formation: results of a controlled therapeutic trial. | 1988 | 3053613 | |
| treatment of campylobacter pylori gastritis: a pilot study using pirenzepine dihydrochloride (gastrozepin) and three formulations of colloidal bismuth subcitrate (de-nol). | antral biopsies were obtained to detect campylobacter pylori infection in 382 patients referred for gastroscopy. one hundred and seventy four patients (46%) were infected. infection was strongly associated with histological gastritis (p less than 0.001), but there was no association between histological antral gastritis and the appearance of the gastric antrum during gastroscopy. because it has been suggested that the lower relapse rate for duodenal ulcer following colloidal bismuth subcitrate ( ... | 1988 | 3054641 |
| campylobacter pylori and gastroduodenal disease: a prospective endoscopic study and comparison of diagnostic tests. | a series of 72 adult patients undergoing 76 upper gastrointestinal (gi) endoscopies because of gi signs or symptoms were studied for the presence of campylobacter pylori by culture, histology, and rapid urease determination of gastric antral biopsy specimens. c. pylori was found by culture or histology in all 10 cases of gastric and duodenal ulcer, and in 77% of endoscopies with histologically proven active gastritis. positive culture for c. pylori was highly correlated pathologically with activ ... | 1988 | 3055942 |
| [campylobacter pylori, ulcer and gastritis: another piece from an old puzzle?]. | 1988 | 3056155 | |
| non-ulcer dyspepsia and short term de-nol therapy: a placebo controlled trial with particular reference to the role of campylobacter pylori. | this double blind randomised study tested the effectiveness of colloidal bismuth subcitrate (de-nol) in non-ulcer dyspepsia (nud) and if any benefit is associated with clearance of campylobacter pylori (c pylori) from the gastric mucosa. sixty six patients with dyspepsic symptoms, normal abdominal ultrasound, and upper gi endoscopy, were randomly allocated to placebo or de-nol for eight weeks. antral biopsies were taken for bacteriological and histological examination, and endoscopies and clinic ... | 1988 | 3058555 |
| [campylobacter pyloridis and its relation to peptic ulcer and chronic gastritis]. | 1988 | 3059039 | |
| [campylobacter pylori and gastric ulcer]. | 1988 | 3059629 | |
| [campylobacter pylori]. | 1988 | 3060270 | |
| [bacteriologic, histologic and serologic studies of the detection of campylobacter pylori in stomach diseases]. | 1988 | 3062918 | |
| [campylobacter pylori. is a routine search for it useful?]. | 1988 | 3063272 | |
| [gastritis, peptic ulcer and campylobacter pylori]. | 1988 | 3064995 | |
| [campylobacter pylori and therapy with bismuth salts]. | 1988 | 3067064 | |
| [campylobacter pylori: a new light on the etiology of chronic gastritis and peptic ulcer?]. | 1988 | 3067265 | |
| campylobacter pylori infection and its relation to chronic gastritis. an endoscopic, bacteriologic, and histomorphologic study. | campylobacter pylori was isolated from 27 of 61 gastric antral biopsy specimens and from 8 of 61 duodenal biopsy specimens. a significant correlation between the occurrence of c. pylori and chronic active gastritis was demonstrated. however, the presence of the bacterium on normal mucosa weakens the theory of c. pylori as a primary causal organism. there was a significant correlation between isolation of c. pylori and erosive lesions in the antral mucosa as diagnosed by endoscopy. no correlation ... | 1988 | 3067331 |
| the protective role of gastric acid. | this paper reviews the relationship between gastric acid secretion and infection and the protective role of gastric acid as a primary bactericidal barrier and modulator of gastrin section. gastric acid is bactericidal at ph 3 or less, but reduction of acidity predisposes to infection with a wide variety of bacteria. bacterial infections or hyperpyrexia may be associated with a marked reduction in gastric acid secretion, and campylobacter pylori has been suggested as one cause of epidemic hypochl ... | 1988 | 3067334 |
| the campylobacter pylori story. | the campylobacter pylori story began before the turn of the century, with early works describing 'spirochaetes' in the gastric mucosa of animals. culture of the organism in 1982 enabled investigators to make sense of the many previous works concerning the microbiology, biochemistry, and histology of the gastric mucosa. whereas some physicians remain skeptical of c. pylori's pathogenic role, those who have studied the new organism believe it is a major gastrointestinal pathogen and see the possib ... | 1988 | 3067335 |
| [what is safe in the treatment of campylobacter pylori-induced gastritis and campylobacter pylori-associated peptic ulcer?]. | 1988 | 3069787 | |
| [the role of campylobacter pyloridis in the etiology and pathogenesis of peptic ulcer]. | 1988 | 3070803 | |
| [detection of campylobacter pylori in gastric biopsies: a comparative study of 4 histopathologic methods and scanning electron microscopy]. | 1988 | 3071267 | |
| does the choice of acute treatment influence future ulcer relapse? | despite the fact that the direct effect of drugs used for healing of ulcers does not last longer than hours, or at most a few days, some clinical studies have indicated that the rate of new ulcers after short courses of treatment depends on the drug used for healing. several studies have now shown that the development of new ulcers in the same, regardless whether active ulcers have been healed with antacids or histamine h2-receptor antagonists. this lack of difference in clinical outcome is very ... | 1988 | 3072664 |
| campylobacter pylori. | worldwide campylobacter pylori is a major cause of active chronic gastritis in man. this curved spiraled microorganism can readily be detected within the mucusgel especially in the antrum, in particular in patients suffering from peptic ulcer disease or non-ulcer dyspepsia, rarely in individuals with normal gastroduodenal mucosa. increasingly arguments are being presented in support of a pathogenetic role of c. pylori in non-ulcer dyspepsia and in peptic ulcer disease. there is a striking discor ... | 1988 | 3072666 |
| campylobacter pyloridis is not responsible for duodenal ulcer formation, result of a controlled therapeutic trial. | 1988 | 3074085 | |
| campylobacter pylori and pernicious anaemia. | 1988 | 2891919 | |
| campylobacter pylori and duodenogastric reflux in peptic ulcer disease and gastritis. | 1988 | 2891956 | |
| campylobacter pylori and development of duodenal ulcer. | 1988 | 2891957 | |
| a decade of campylobacter pylori. | 1988 | 2893219 | |
| campylobacter pylori and ulcer recurrence. | 1988 | 2894530 | |
| one-minute endoscopy room test for campylobacter pylori. | 1988 | 2895232 | |
| campylobacter pylori and protein losing enteropathy in children. | 1988 | 2895370 | |
| campylobacter pylori urease: a new serological test. | 1988 | 2896815 | |
| campylobacter pylori in patients with gastritis, peptic ulcer, and carcinoma of the stomach in lanzhou, china. | 1988 | 2896897 | |
| [ofloxacin in therapy of "resistant" duodenal ulcer. a pilot study]. | since its description in 1983 campylobacter pylori (c.p.) has been discussed as possible pathogenic factor at least in duodenal ulcer disease. the therapeutic combination of h2-receptor-antagonists (= h2-ra) and ofloxacin (tarivid) has shown to heal resistant duodenal ulcers in some preliminary cases, which did not respond to a three month standard dosage treatment with h2-ra. this paper describes the results of twelve patients whose duodenal ulcers were resistant to h2-ra. c.p. was detected in ... | 1988 | 2900584 |
| campylobacter pylori in abattoir workers: is it a zoonosis? | sera from 98 abattoir workers were tested for igg to campylobacter pylori, c jejuni, and klebsiella. clerical workers had significantly lower c pylori and c jejuni igg titres than any of the groups in direct contact with freshly cut animal parts. no difference was found for antibodies to klebsiella. 28 non-clerical workers with high-titre c pylori igg consented to upper gastrointestinal endoscopy. c pylori associated gastritis was found in all 28, and four weeks of colloidal bismuth subcitrate ( ... | 1988 | 2901576 |
| effect of acid inhibition on campylobacter pylori. | campylobacter pylori (cp) infection has been demonstrated in 59 of 65 (91%) patients with gastric or duodenal ulcer. patients were included in a double blind study with ranitidine or famotidine for 4-8 weeks. after therapy, all gastric and duodenal ulcers healed but cp remained in all previously infected patients, associated with chronic inflammation of gastric mucosa. our results suggest that h2 antagonists do not have any effect either on cp infection or on concurrent chronic gastritis. at the ... | 1988 | 2901784 |
| is campylobacter pylori a zoonosis? | 1988 | 2902418 | |
| is campylobacter pylori a zoonosis? | 1988 | 2903369 | |
| the pathophysiological and pharmacological basis of peptic ulcer therapy. | both genetic and nongenetic factors predispose to ulcer diathesis. at the mucosal level ulcers result from an imbalance between aggressive factors and mucosal defense. ulcer therapy reduces aggressive forces, bolsters defense, or both. gastric acid, the major aggressive factor, may have its secretion inhibited or it may be partially neutralized by antacids. h2 receptor antagonists competitively block histamine occupancy of h2 receptors on parietal cells, thereby preventing stimulation of adenyla ... | 1988 | 2903542 |
| "eradication" of campylobacter pylori: are we being misled? | 1988 | 2903967 | |
| prospective double-blind trial of duodenal ulcer relapse after eradication of campylobacter pylori. | 100 consecutive patients with both duodenal ulcer and campylobacter pylori infection were followed up to see whether eradication of c pylori affected ulcer healing or relapse. patients were randomly assigned to 8 weeks of treatment with cimetidine or colloidal bismuth subcitrate (cbs), with tinidazole or placebo being given concurrently from days 1 to 10, inclusive. endoscopy, biopsy, and culture were done at entry, in weeks 10, 22, 34, and 62, and whenever symptoms recurred. there was no mainte ... | 1988 | 2904568 |
| duodenal ulcer, campylobacter pylori, and the "leaking roof" concept. | 1988 | 2904580 | |
| future trends in the management of peptic ulcer disease. | future trends in the short- and long-term management of peptic ulcer disease are considered. the present state of development of pharmacologic agents for the short-term healing of duodenal and gastric ulcer is impressive, and high rates of healing with rapid symptom relief can be safely achieved with several agents. the problem of ulcer recurrence has not been solved, although new work concerning the role of campylobacter pylori holds promise. the mortality of acute gastrointestinal haemorrhage ... | 1988 | 2906462 |
| comparison between medical and elective surgical treatment of peptic ulcers. | during the last decades we have learnt how to treat chronic peptic ulcer disease both with surgery (mainly vagotomy) and with long term medical treatment (mainly h2-receptor antagonists) with great success and safety. although much attention has lately been given to 'cytoprotective' agents, it is still too early to regard them as alternatives to acid reducing treatment in the long term management of severe peptic ulcer disease. the importance of campylobacter pylori for long term outcome is toda ... | 1988 | 2907681 |
| campylobacter pylori: fact or fancy? | 1988 | 3335291 | |
| campylobacter pyloridis-associated chronic active antral gastritis. a prospective study of its prevalence and the effects of antibacterial and antiulcer treatment. | to determine the clinical importance of campylobacter pyloridis infection, its association with gastric inflammation, and the response to drug therapy, patients with a duodenal or gastric ulcer (n = 63), patients with nonulcer dyspepsia (n = 240), and asymptomatic volunteers (n = 34) were studied. in a prospective longitudinal study, the type, intensity, and distribution of inflammation in antral biopsy specimens were correlated with the presence of c. pyloridis. campylobacter pyloridis was cult ... | 1988 | 3335295 |
| carbon-14 urea breath test for the diagnosis of campylobacter pylori associated gastritis. | urease in the human gastric mucosa is a marker for infection with campylobacter pylori (cp), an organism suspected of causing chronic gastritis and peptic ulceration. to detect gastric urease, we examined 32 patients who were being evaluated for possible peptic ulcer disease. fasting patients were given 10 microci (370 kbq) of 14c-labeled urea. breath samples were collected in hyamine at intervals between 1 and 30 min. the amount of 14c collected at these times was expressed as: body weight x (% ... | 1988 | 3335917 |
| non-ulcer dyspepsia: a readily-treatable disorder with no relation to campylobacter pylori. | 1988 | 3336320 | |
| campylobacter pylori gastritis not related to periodontal disease. | 1988 | 3341346 | |
| campylobacter pylori and antral gastritis in children. | 1988 | 3341347 | |
| characterization of an unclassified microaerophilic bacterium associated with gastroenteritis. | four isolates of an unclassified microaerophilic bacterium resembling campylobacter species were characterized by growth requirements, microscopic examination, biochemical characteristics, antimicrobial susceptibility tests, and protein profile analysis. the unclassified isolates were differentiated from campylobacter jejuni, campylobacter coli, campylobacter fetus subsp. fetus, campylobacter laridis, campylobacter pylori, and an ovine isolate. the bacterium was fusiform shaped with a corrugated ... | 1988 | 3343302 |
| urease activity of campylobacter pylori. | 1988 | 3343372 | |
| campylobacter associated gastritis in patients with non-ulcer dyspepsia. | gastric biopsy specimens from 109 patients with non-ulcer dyspepsia were retrospectively examined. sixty one patients had gastritis and there was a strong correlation with the presence of campylobacter pyloridis. ninety eight per cent were positive in large numbers for c pyloridis by histological examination or by culture, or both. of 48 patients with normal histological results, 21 had evidence of c pyloridis by histological examination or culture, or both, but in small numbers. it is concluded ... | 1988 | 3343382 |
| gastritis. bringing this enigma into sharper focus. | type a and type b (probably campylobacter pylori) gastritis are distinct clinical (but largely asymptomatic) and histologic entities that can be separated from other disease processes. while the association of c pylori gastritis with peptic ulcer disease is well documented, no causal relationship has been established. acute inflammatory conditions of the stomach secondary to other disease processes, macroscopic abnormalities of the gastric mucosa, and sometimes poorly characterized epigastric pa ... | 1988 | 3344261 |
| histological gastritis in duodenal ulcer: relationship to campylobacter pylori and effect of ulcer therapy. | antral gastritis has been described in up to 100% of patients with duodenal ulcer disease, and recently has been associated with campylobacter pylori infection. the present study examines the effect of two ulcer therapies (cimetidine and colloidal bismuth subcitrate) on this lesion. sixty-four consecutive patients with endoscopically diagnosed duodenal ulcer were randomized to treatment with either agent upon entry into the study. antral biopsies were taken at both initial and final endoscopies ... | 1988 | 3344730 |
| campylobacter pylori is associated with chronic gastritis but not with active peptic ulcer disease. | campylobacter pylori is supposed to be involved in the pathogenesis of gastroduodenal peptic ulcer diseases and chronic gastritis. in order to study whether the campylobacter pylori in the stomach of peptic ulcer patients is related to ulcer itself or to a co-existing chronic gastritis, we examined the frequency of the bacteria in giemsa stained histological sections of biopsy specimens from a series of patients with active peptic ulcer and from series of non-ulcer control subjects. we found no ... | 1988 | 3345253 |
| acid secretion and serum gastrin levels in individuals with campylobacter pylori. | campylobacter pylori may cause gastritis and has been proposed as an etiologic factor in the development of peptic ulcer. however, it may be an acid-sensitive microbe and before it can be implicated in the pathogenesis of peptic ulcer, it should be consistently found in ulcer patients with normal acid secretion. thirty-six patients with c. pylori by warthin-starry stain underwent gastric analysis; 25 were normochlorhydric and 11 hypochlorhydric. ulcers were present in 19 normochlorhydric patient ... | 1988 | 3345893 |
| effect of age on the frequency of active campylobacter pylori infection diagnosed by the [13c]urea breath test in normal subjects and patients with peptic ulcer disease. | we studied the frequency of active campylobacter pylori infection in persons from north america (n = 53) or the people's republic of china (n = 15) who had no gastrointestinal symptoms or a history of ulcer disease and in patients with active or recently active duodenal or gastric ulcers diagnosed by endoscopy (n = 105). c. pylori infection was significantly (p less than .001) more frequent in patients with peptic ulcers (88%) than in the normal group (45%). an age-related increase in the freque ... | 1988 | 3346569 |
| demonstration of plasmids in campylobacter pylori. | 1988 | 3346576 | |
| bacteria of the gastric antrum and their relation to chronic gastritis. | biopsy samples from the gastric antrum were taken from 61 patients. on bacteriological culture, campylobacter pylori was isolated in 27 subjects. thirty-four patients had chronic gastritis, as seen in routine-stained histological sections. by means of the may-grünwald-giemsa (mgg) staining technique, bacteria were demonstrated in sections from 26 subjects. of these, 22 had gastritis histologically. in 13 subjects, structures similar to campylobacter pylori were found in mgg-stained sections, 11 ... | 1988 | 3348915 |
| [campylobacter pylori--status of knowledge from the morphologic viewpoint]. | the morphological alterations of the gastric mucosa due to campylobacter pylori (c.p.) were examined with high resolution light microscopy, scanning electron microscopy, and transmission electron microscopy. on the ultrastructural level the number and distribution of bacteria were correlated to the extent of the inflammatory resp. degenerative epithelial alterations. intestinal metaplasia was always free of bacteria, even with strong colonization of the residual mucosa. in comparison to the gast ... | 1988 | 3352454 |
| campylobacter pylori infection in patients undergoing endoscopy in fiji. | forty two patients (20 fijian and 22 indian) presenting for endoscopy at the colonial war memorial hospital, suva, fiji, were biopsied to detect campylobacter pylori infection. detection of the organism's urease activity in biopsy material or seeing the organism in warthin-starry silver stained histology sections were used to diagnose infection. thirty-nine patients (93%) were infected; 19 of 20 fijians (95%) and 20 of 22 indians (91%). of the 39 infected patients, 37 (95%) had chronic active ga ... | 1988 | 3353031 |
| [the occurrence of campylobacter pyloridis in patients with upper gastrointestinal dyspepsia]. | 1988 | 3354080 | |
| effect of treatment on campylobacter pylori in peptic disease: a randomised prospective trial. | this study investigated the effect of colloidal bismuth subcitrate and cimetidine on campylobacter pylori in peptic disease. in 74% of 135 patients with peptic disease diagnosed at endoscopy c pylori was detected before treatment. compared with cimetidine, colloidal bismuth subcitrate significantly decreased the incidence of c pylori after six weeks of treatment (p less than 0.001). in the colloidal bismuth subcitrate group, subsequent healing of the lesion was correlated with the clearance of c ... | 1988 | 3356357 |
| campylobacter pylori-associated gastritis. is antibiotic therapy now justified? | campylobacter pylori clearly is a common inhabitant of the human stomach and is associated with nonautoimmune antral gastritis, chronic peptic ulceration, and non-ulcer dyspepsia. this editorial reviews whether therapy is now justified for patients with this infection. the criteria that must be fulfilled before an association can be considered to be of etiologic significance are discussed in relation to c. pylori infection and clinical disease. an overview of therapeutic approaches to eradicate ... | 1988 | 3356873 |
| antibody response to campylobacter pylori in an ethnic group lacking peptic ulceration. | the association between duodenal ulcer, gastritis and gastroduodenal colonization with campylobacter pylori suggests a causal role for this newly described bacterium. in an attempt to challenge the verity of this association we studied a group of people in whom duodenal ulcer is apparently absent. serological evidence of infection was sought with a sensitive, specific elisa assay for c. pylori specific igg and was compared with results from control sera from teenagers referred for respiratory vi ... | 1988 | 3363304 |
| should we now, routinely, be examining gastric biopsies for campylobacter pylori? gastric mucosal biopsy: an essential investigation in patients with dyspepsia. | 1988 | 3364408 | |
| should i search for campylobacter pylori in my patients? much ado about not much? | 1988 | 3364409 | |
| type b gastritis, aging, and campylobacter pylori. | 1988 | 3365072 | |
| gastritis and campylobacter pylori in healthy, asymptomatic volunteers. | a high prevalence of histologic gastritis in asymptomatic individuals has been reported in the literature. the studies have been poorly controlled for gastritis risk factors. we evaluated 20 healthy, asymptomatic volunteers free of known risk factors for gastritis with endoscopy, mucosal biopsy, culture, and gastric ph determination. the prevalence of gastritis on histologic examination was found to be 20%. there was no relationship between the presence of macroscopic findings at endoscopy and t ... | 1988 | 3365082 |
| nonsteroidal anti-inflammatory drug-induced dyspepsia--is campylobacter pyloridis implicated? | an endoscopic study of 34 patients with rheumatic diseases taking nonsteroidal anti-inflammatory drugs (nsaids) was undertaken to evaluate whether dyspepsia was associated with campylobacter pyloridis. twenty-two patients had an indication for upper gastrointestinal endoscopy and 12 patients were asymptomatic volunteers. dyspepsia did not correlate with the macroscopic findings at endoscopy, the presence of histological gastritis or the presence of c. pyloridis. however, this study did demonstra ... | 1988 | 3365528 |
| campylobacter pylori associated gastritis in patients with rheumatoid arthritis taking nonsteroidal anti-inflammatory drugs. | fifty-two patients with rheumatoid arthritis taking nonsteroidal anti-inflammatory drugs were studied in order to assess the carriage rate of campylobacter pylori (c. pylori) with reference to dyspeptic symptoms, endoscopic appearance and antral histology. all patients were interviewed using a standard gastrointestinal symptom scoring questionnaire and underwent endoscopy at which two antral biopsies were obtained. sections were examined for the presence and severity of gastritis and of c. pylor ... | 1988 | 3365529 |
| [campylobacter pylori and gastric acidity]. | 1988 | 3366322 | |
| campylobacter pylori infection in children. | two children with antral nodular hyperplasia and active chronic gastritis associated with campylobacter pylori are reported. the organism was observed by scanning electron microscopy and cultured from antral biopsies obtained at endoscopy from each patient. anti-c. pylori igg was detected in high titre in both patients together with serum igm in one patient. treatment with ampicillin and a bismuth preparation resulted in total resolution of symptoms. repeated endoscopy with antral biopsy and cul ... | 1988 | 3367059 |
| prevalence of campylobacter pylori and association with antral mucosal histology in subjects with and without upper gastrointestinal symptoms. | to determine the incidence and significance of campylobacter pylori in patients undergoing routine endoscopic evaluation at this hospital, biopsies of the antrum were obtained from 132 patients and from 15 asymptomatic volunteers. specimens were cultured and silver stained for the presence of c. pylori and were examined histologically. c. pylori was detected in 67 (51%) patients and two (13%) volunteers (p = 0.006). in patients, c. pylori was found significantly more often when the mucosa was in ... | 1988 | 3371136 |
| campylobacter pylori gastritis in the acquired immunodeficiency syndrome. | campylobacter pylori has been associated with gastritis, duodenitis, and duodenal ulceration in the immunocompetent individual. it has been described within the superficial mucus layer, in interepithelial junctions, and occasionally in the microcanaliculi of epithelial cells, but never in the lamina propria. we describe a case of invasive c. pylori in a patient with the acquired immunodeficiency syndrome and discuss its clinical presentation and histopathological findings. | 1988 | 3371616 |
| gastric and esophageal campylobacter pylori in patients with barrett's esophagus. | campylobacter pylori organisms were found with similar frequency in the stomachs of patients with barrett's esophagus and in age- and sex-matched controls (10 of 26 vs. 11 of 26). campylobacter pylori was also observed in esophageal barrett's mucosa in some patients with gastric c. pylori, but not when gastric infection was absent (4 of 10 vs. 0 of 16). campylobacter pylori was not detected in esophageal squamous mucosa from patients with barrett's esophagus or in 25 non-barrett's patients with ... | 1988 | 3371618 |
| campylobacter pylori and gastric antral intestinal metaplasia. | 1988 | 3371622 |