Publications
| Title | Abstract | Year(sorted ascending) Filter | PMID Filter |
|---|
| appropriate acid suppression in the treatment of acid-related conditions. | gastric acid is involved in the pathogenesis of duodenal ulcer, gastric ulcer and gastro-oesophageal reflux disease. although levels of acid secretion may not be abnormally high in patients with these conditions, pharmacological suppression of acid secretion is associated with healing of mucosal defects and maintenance of healing in the long term. in the case of duodenal ulcer, this was demonstrated before the understanding of the importance of helicobacter pylori infection. there is a considera ... | 1994 | 7972343 |
| gastritides, gastropathies, and polyps unique to the stomach. | this article discusses a variety of non-neoplastic conditions that involve the stomach. nonspecific gastritis and helicobacter gastritis are discussed in detail as are other infectious diseases and the forms of hypertrophic gastritis. non-neoplastic polyps unique to the stomach, hyperplastic polyps, and fundic gland polyps are also covered. | 1994 | 7972709 |
| gastrospirillum hominis in acute gastric erosion. | gastrospirillum hominis is a spirochete that has been described in association with chronic gastritis and duodenal ulcers. we report the case of a patient having acute gastric erosion in whom biopsy showed abundant organisms. the erosion resolved while the patient was receiving sucralfate and omeprazole therapy. we discuss the histopathology and mode of transmission of g hominis, along with the role of antibiotic therapy. | 1994 | 7973903 |
| gastrointestinal bleeding in stroke. | patients with ischemic or hemorrhagic stroke are at risk for systemic complications. the reasons why gastrointestinal bleeding occurs after stroke are unknown and have intuitively been attributed to stress ulcers. no study to date has addressed causes of gastrointestinal hemorrhage in stroke. | 1994 | 7974536 |
| [helicobacter pyloris: quo vadis?]. | 1994 | 7975666 | |
| [helicobacter pylori: pathogens, pathomechanisms and epidemiology]. | h. pylori organisms are microaerophilic gram-negative curved or spiral bacteria that live in the mucus layer of the gastric epithelium. since the discovery of h. pylori similar organisms have been found in humans (h. cinaedi, h. fennelliae, h. heilmanii) and animals. h. pylori causes chronic active gastritis and is an important factor in the development of peptic ulceration and gastric cancer. narrow host range, tissue specificity and chronic inflammation are characteristic features. putative vi ... | 1994 | 7975667 |
| [current therapy of helicobacter pylori-induced chronic gastritis and/or peptic ulcer]. | helicobacter pylori (h.p.) induced chronic gastritis cannot at present be considered as an absolute indication for eradication therapy of h.p. clinical trials with antimicrobial compounds, mainly bismuth, have not convincingly demonstrated appreciable symptomatic benefit for patients with h.p.-positive gastritis and non-ulcer dyspepsia. however, new results indicate that possibly a longer follow-up period may be necessary to prove a symptomatic improvement after h.p. eradication in patients with ... | 1994 | 7975668 |
| [helicobacter pylori and the mouth cavity--overview and perspectives]. | in view of the possibility of reinfection after successful treatment and the pitfalls in eradicating helicobacter pylori (h. pylori) from gastric mucosa, it is of great interest to identify natural reservoirs for this organism, other than the stomach. this review discusses the results of investigations as to whether h. pylori can be harboured in the microaerobic environment of dental plaques in saliva. only few data are available on the prevalence of h. pylori in the mouth. data from conventiona ... | 1994 | 7975669 |
| [therapeutic relevance of the classification of gastritis]. | the histological gastritis classification according to the sydney system is a standardisation of different classification systems used so far. it is based on the principles of etiology, topography and course of the disease, as well as morphological changes of the gastric mucosa in gastritis. histological examination represents the practical gold standard of helicobacter pylori detection and can be used on routinely formaldehyde-fixed biopsy material and hematoxylin-eosin stained slides. histolog ... | 1994 | 7975670 |
| [helicobacter pylori, a risk factor for stomach cancer and primary stomach lymphoma?]. | helicobacter pylori has been implicated as a possible etiologic factor in gastric cancer. recent epidemiologic studies, in fact, have demonstrated that there is a significantly higher prevalence of h. pylori in patients with gastric cancer than in matched controls. in addition to these and other epidemiologic findings, there is evidence that several of the pathogenic consequences of h. pylori infection may be relevant to carcinogenesis. in accordance with the theory that the etiology of gastric ... | 1994 | 7975671 |
| [diagnosis of helicobacter pylori infection]. | a number of direct (histology, specific culture) and indirect (serology, urease test, breath test) diagnostic tests for helicobacter pylori (h. pylori) are available. the gold standard for h. pylori presence or absence is still histology (tissue stained by giemsa) without or in combination with specific culture. for routine practice a combination of histology (two antral and two body biopsies) and the urease test (one antral and one body biopsy specimen) is recommended in patients undergoing upp ... | 1994 | 7975672 |
| [24-hour gastric ph profile with 2 x 20 mg and 2 x 40 mg omeprazole in patients with helicobacter pylori-associated gastroduodenal ulcer disease]. | fifty patients suffering from helicobacter pylori associated duodenal (n = 25) or gastric ulcer disease (n = 25) were randomly treated with either omeprazole 20 mg b.i.d. (n = 25) or 40 mg b.i.d. (n = 25). from day 8 to 9, a 24-hour gastric ph measurement was performed in all patients. patients with duodenal ulcer disease treated with 40 mg or 80 mg omeprazole demonstrated similar gastric ph patterns without statistically significant differences with regard to the mean (5.10 versus 5.17, p = 0.6 ... | 1994 | 7975785 |
| [h. pylori and chronic duodenal ulcer: eradication therapy with ranitidine-antibiotic combination and follow-up of ulcer disease]. | 1994 | 7975793 | |
| helicobacter pylori and chronic antral gastritis in elderly patients. | fifty-five elderly patients with chronic antral gastritis (cag) were studied to assess the relationship between helicobacter pylori (h. pylori) status and cag subtypes as specified in the sydney system for gastritis classification. twenty-eight patients (51%) were h. pylori positive and 27 (49%) h. pylori negative. h. pylori-positive patients had a significantly greater association with features of severe active cag (chronic inflammation and polymorph activity) than h. pylori-negative patients. ... | 1994 | 7976771 |
| the epidemiology of helicobacter pylori in peruvian children between 6 and 30 months of age. | cross-sectional studies of children in developing countries show a high prevalence of helicobacter pylori infection at 6 months of age, but a decrease in the prevalence of infection between 1 and 5 yr of age. the decrease suggests a loss or clearance of infection, an uncommon finding in adults. our objective in this study was to determine the longitudinal persistence of h. pylori infection in young children. | 1994 | 7977241 |
| seroprevalence of helicobacter pylori infection in children of low socioeconomic level in belo horizonte, brazil. | to study the epidemiology of helicobacter pylori infection in children of low socioeconomic level in belo horizonte, brazil. | 1994 | 7977242 |
| effects of helicobacter pylori infection and gastritis on gastric alcohol dehydrogenase activity. | sex and age differences in gastric alcohol dehydrogenase activity in relation to abnormalities of gastric histology and helicobacter pylori infection were determined in 63 patients (32 men and 31 women) undergoing upper endoscopy for gastrointestinal symptoms. no sex difference was found in gastric alcohol dehydrogenase activity. males older than 50 years had lower enzyme activity than younger males. patients with h. pylori and/or moderate to severe chronic and acute inflammation and epithelial ... | 1994 | 7978087 |
| helicobacter pylori, active chronic antral gastritis, and gastrointestinal symptoms in alcoholics. | the frequency of helocobacter pylori (hp.) infection and active chronic antral gastritis among people with excessive alcohol consumption is not known. a high alcohol intake regularly causes acute gastroduodenitis. in this study, the prevalence of hp. infection and active chronic antral gastritis in alcoholics compared with nonalcoholic controls was studied. further, the frequency of gastrointestinal symptoms was registered. diagnostic methods for hp. were compared. twenty-four alcoholics admitte ... | 1994 | 7978100 |
| tight binding of clarithromycin, its 14-(r)-hydroxy metabolite, and erythromycin to helicobacter pylori ribosomes. | clarithromycin is a recently approved macrolide with improved pharmacokinetics, antibacterial activity, and efficacy in treating bacterial infections including those caused by helicobacter pylori, an agent implicated in various forms of gastric disease. we successfully isolated ribosomes from h. pylori and present the results of a study of their interaction with macrolides. kinetic data were obtained by using 14c-labeled macrolides to probe the ribosomal binding site. clarithromycin, its parent ... | 1994 | 7979278 |
| pyruvate metabolism in helicobacter pylori. | the metabolism of pyruvate by helicobacter pylori was investigated employing one- and two-dimensional 1h and 13c nuclear magnetic resonance spectroscopy. generation of pyruvate from l-serine in incubations with whole cell lysates indicated the presence of serine dehydratase activity in the bacterium. pyruvate was formed also in cell suspensions and lysates from phosphoenol pyruvate. metabolically competent cells incubated aerobically with pyruvate yielded alanine, lactate, acetate, formate, and ... | 1994 | 7979873 |
| touch cytology. a quick, simple, sensitive screening test in the diagnosis of infections of the gastrointestinal mucosa. | to assess the role of touch cytology (imprint from endoscopic biopsy specimens) in the diagnosis of mucosal infections of the gastrointestinal tract, we reviewed all records and specimens of patients seen during a 30-month period. touch cytology was performed by rolling biopsy specimens on glass slides. after air fixation, a rapid staining method similar to may-grünwald-giemsa was used. the following infections and pathogens were diagnosed (in decreasing order of frequency): helicobacter pylori ... | 1994 | 7979897 |
| langerhans cell histiocytosis of the stomach. a cause of granulomatous gastritis and gastric polyposis. | langerhans cell histiocytosis (lch) of the alimentary tract is a rare condition, and lch of the stomach especially so. we report a histologically and endoscopically unusual case of gastric lch, the diagnosis of which was based on gastric mucosal biopsy specimens from an adolescent with multi-organ lch and striking gastric polyposis. microscopically, the gastric mucosa was expanded by discrete granulomatous microaggregates of langerhans cells, resulting in a close resemblance to other, more commo ... | 1994 | 7979922 |
| helicobacter pylori. | 1994 | 7982436 | |
| seroprevalence of helicobacter pylori infection is not increased in pediatric inflammatory arthritides. | to determine the seroprevalence of h. pylori infection among children with inflammatory arthritides receiving antiinflammatory drug therapy. | 1994 | 7983663 |
| [usefulness of the breath test with urea-13c in the diagnosis of helicobacter pylori infection]. | many diagnostic methods of variable diagnostic profitability are available to detect infection by helicobacter pylori. the aim of this study was to evaluate the usefulness of the european method of the breath test with urea-13c and compare the results with common histologic methods, the rapid urease test and serology. | 1994 | 7983899 |
| gastroenteritis associated with helicobacter pullorum. | 1994 | 7983966 | |
| the human gastric pathogen helicobacter pylori has a gene encoding an enzyme first classified as a mucinase in vibrio cholerae. | the human bacterial pathogen helicobacter pylori has been suggested to be the causative agent of the most common chronic infection of man. since its first isolation in 1982, h. pylori has been associated with gastric and duodenal ulcer disease, and more recently, gastric cancer. the proteolytic digestion of gastric mucus by this microorganism has been suggested as an important mechanism by which its pathogenicity is at least partly exerted. here we report the detection of protease activity in h. ... | 1994 | 7984089 |
| [h. pylori and gastroduodenal pathology in 1994. a change of medical attitude to peptic ulcer]. | 1994 | 7986616 | |
| review article: treatments that impact favourably upon the eradication of helicobacter pylori and ulcer recurrence. | healing the gastroduodenal mucosa through helicobacter pylori eradication leads to a dramatic reduction in gastroduodenal ulcer relapse. eradication of h. pylori proves to be difficult. although the organism is sensitive to many antibiotics in vitro, the in vivo eradicating efficacy is often disappointing. this overview summarizes the most commonly used currently-available eradication schemes and the consequences of successful eradication in peptic ulcer disease. | 1994 | 7986961 |
| review article: helicobacter pylori eradication--understandable caution but no excuse for inertia. | the long-term management of patients with peptic ulcer disease is unsatisfactory, as judged by the persistently high levels of haemorrhage, perforation and death from this condition in western countries. although ulcer recurrence and complications can be prevented, many patients with peptic ulcer disease fail to receive the benefits of modern therapeutic regimens. in recent years, eradication of helicobactor pylori has been promoted as a 'cure' for peptic ulcer disease and, while such claims are ... | 1994 | 7986962 |
| clarithromycin-amoxycillin therapy for helicobacter pylori infection. | more convenient therapies are needed to treat helicobacter pylori infection successfully. clarithromycin and amoxycillin are effective against h. pylori both in vivo and in vitro. recent success with a high dose amoxycillin-metronidazole combination therapy led us to evaluate clarithromycin-amoxycillin dual therapy for h. pylori infection. | 1994 | 7986970 |
| omeprazole, bismuth and clarithromycin in the sequential treatment of helicobacter pylori infection. | to assess the therapeutic potential of clarithromycin, a new macrolide with high anti-helicobacter pylori activity, given with bismuth salts and omeprazole in different regimens aimed at simplifying the treatment of h. pylori-related gastritis. | 1994 | 7986974 |
| helicobacter pylori infection in childhood: risk factors and effect on growth. | to investigate the current prevalence of helicobacter pylori infection in childhood, the risk factors for infection, and the effect of infection on growth in preadolescent schoolchildren. | 1994 | 7987103 |
| genetic diversity of the helicobacter pylori haemagglutinin/protease (hap) gene. | helicobacter pylori has been associated with the etiology of gastritis, gastric and duodenal ulcers and gastric cancer in man. in this study we confirm the presence of the haemagglutinin/protease (hap) gene in ten geographically diverse strains of h. pylori, but the southern analysis of genomic dna suggested that the location of the hap gene was different in each strain. we suggest that the hap gene is subject to a high rate of mutation and subsequent polymorphisms by virtue of a mobile, genetic ... | 1994 | 7988885 |
| innovations in gastrointestinal diseases. | 1994 | 7991485 | |
| low incidence of clinically significant gastroduodenal pathology despite a high incidence of helicobacter pylori infection. | gastric helicobacter pylori infection is common throughout the tropics yet does not always correlate with the incidence of serious upper gastrointestinal pathology. in a consecutive series of 213 patients examined by gastroscopy for dyspepsia in northern nigeria, 176 (92%) of 193 with acceptable biopsies had gastritis. only 16 (8%) had a histologically normal gastric mucosa. h. pylori was present in 161 of 192 patients (84%); 31/41 (75%) with chronic gastritis and 130/135 (96%) with active gastr ... | 1994 | 7992342 |
| a study of helicobacter-pylori in 100 pediatric patients from the tri-state area. | helicobacter pylori (hp) is a newly discovered pathogen implicated in the pathophysiology of peptic ulcer disease. the aim of this study was to review all pediatric patients who were evaluated by upper endoscopy through the pediatric gastroenterology service at the marshall university school of medicine between july 1990 and march 1993. a total of 100 charts were retrospectively reviewed. hp was diagnosed by clo-test and confirmed histologically. results showed that the major presenting symptom ... | 1994 | 7992484 |
| awakenings to the pathogenicity of urease and the requirement for continuous long term therapy. | urease is an enzyme found in plants and bacteria, but not mammals. it catalyzes the conversion of urea to carbon dioxide and ammonia. ammonia shortens the life span of cells; and higher concentrations cause tissue necrosis and cytolysis. twenty percent of total body urea is converted to ammonia by bacterial urease in the colon. small injections of urease immunize animals by producing antiurease, a gamma globulin, which inactivates urease. immunization eliminates the colonic conversion of urea to ... | 1994 | 7993980 |
| ethanol metabolism in the gastrointestinal tract and its possible consequences. | ethanol is oxidised not only in the liver, but also in the gastrointestinal tract. although this ethanol metabolism is less than that of the liver, it has some important relevance with respect to the first pass metabolism of alcohol and to ethanol induced tissue toxicity. in the gastrointestinal tract, ethanol can be metabolised not only in the mucosal cell via alcohol dehydrogenase (adh) and microsomal ethanol oxidising system (meos), but also in a great variety of bacteria. depending on the ga ... | 1994 | 8974330 |
| [ulcer disease and helicobacter pylori]. | 1994 | 7940444 | |
| [10 days of triple treatment of helicobacter pylori infection and peptic ulcer. status after treatment of 4 weeks and of one year]. | we treated 143 helicobacter pylori positive ulcer patients (duodenal ulcer 81, gastric ulcer 24, pyloric ulcer nine, previous duodenal ulcer 27, others two) with a ten days regimen of bismuth subnitrate, oxytetracycline and metronidazole. four weeks after cessation of treatment the 14c-urea breath test showed that the infection was eradicated in 133 out of 140 patients (95%). the ulcer had healed in 129 (97%) of the h. pylori negative patients. status at one year after treatment showed that 121 ... | 1994 | 7940447 |
| [a norwegian conference on updating of the treatment of helicobacter pylori in peptic ulcer. interessegruppe for gastroenterologi]. | recent data on peptic ulcer and helicobacter pylori colonization of the ventricle were discussed. agreement was reached to re-adjust the pharmacological treatment of this condition. all patients for whom antibiotic therapy is considered should be examined by gastroscope. the bacterial agent should be proved by at least one out of several available methods. two different established regimens are prescribed, either triple therapy with bismuth, metronidazole and tetracycline or double treatment wit ... | 1994 | 7940453 |
| [infections of the upper gastrointestinal tract--more than helicobacter pylori?]. | helicobacter pylori infection is a problem frequently handled by the gastroenterologist, but many other infections may cause symptoms and pathological findings which is of great significance for the endoscopist to recognize. candida infection is the most common infection in the oesophagus. it may signal an underlying disease like diabetes. when the patient enters norway from an area where tuberculosis is endemic it is necessary to be aware of possible abdominal tuberculosis. immuno-compromised p ... | 1994 | 7940454 |
| incidence of helicobacter pylori associated gastritis in the urban population from india. | endoscopic gastric biopsies from 109 patients showing chronic active gastritis on histology were analysed to detect helicobacter pylori by gimenez technique. incidence of h. pylori positivity was found to be 50.4%. the organisms were detected mainly above the epithelial surface and in the crypts. all the patients were residents from bombay or its suburbs. | 1994 | 7941008 |
| gastric adenocarcinoma and helicobacter pylori infection. | 1994 | 7941511 | |
| highly significant change of the clinical course of relapsing and complicated peptic ulcer disease after cure of helicobacter pylori infection. | the aim of the present study was to compare the rates of peptic ulcer recurrence and of complications in a cohort of 190 patients, before and up to 4 yr after eradication of helicobacter pylori infection. | 1994 | 7942667 |
| omeprazole 40 mg o.m. combined with amoxycillin alone or with amoxycillin and metronidazole in the eradication of helicobacter pylori. | our objectives were to determine the effect of dual therapy with omeprazole and amoxicillin and of triple therapy with omeprazole, amoxicillin, and metronidazole in the eradication of helicobacter pylori (hp) and to study the long-term results of eradication in these patients. | 1994 | 7942668 |
| long-term follow-up and serological study after triple therapy of helicobacter pylori-associated duodenal ulcer. | the purpose of this study was to monitor the change of igg antibodies to helicobacter pylori during the course of a long-term follow-up after the eradication of h. pylori, as well as to evaluate the efficacy of triple therapy in preventing the recurrence of duodenal ulcer (du). | 1994 | 7942669 |
| alterations in upper gastrointestinal motility in helicobacter pylori-positive nonulcer dyspepsia. | to study the association between gastrointestinal motility and helicobacter pylori (hp) among patients with nonulcer dyspepsia (nud). | 1994 | 7942670 |
| bacterial mucosal infiltration in helicobacter pylori-associated gastritis: histological and clinical consequences. | we wished to demonstrate that gastric epithelial cells infiltration by hp is associated with the active inflammatory response and the severity of gastritis in the gastric antrum of patients harboring the bacterium. | 1994 | 7942671 |
| association of helicobacter pylori with gastric cancer and observations on the detection of this bacterium in gastric cancer cases. | the aim of the present study was to assess the prevalence of helicobacter pylori infection in chinese patients with advanced gastric cancer, to compare this with a matched control population, and to identify factors that may effect the detection of h. pylori in patients with gastric cancer. | 1994 | 7942672 |
| occupational exposure to helicobacter pylori for the endoscopy professional: a sera epidemiological study. | the purpose of this prospective study was to determine if medical and nursing staff in the united states who are regularly involved in endoscopic procedures are at an increased risk of acquiring helicobacter pylori infection. | 1994 | 7942723 |
| randomized study comparing 1 with 2 weeks of quadruple therapy for eradicating helicobacter pylori. | we investigated whether the recommended 2-wk triple therapy for eradicating helicobacter pylori could be reduced to 1 wk, and thus we tried to determine the optimal treatment duration for triple therapy. | 1994 | 7942724 |
| digestive health initiative launches with the helicobacter pylori education program. | 1994 | 7942753 | |
| roxithromycin in the eradication of helicobacter pylori. | 1994 | 7942758 | |
| chronic active hepatitis in mice caused by helicobacter hepaticus. | helicobacter infections cause chronic gastroenteritis in humans and several animal species. we recently discovered a new helicobacter (h. hepaticus) that is the etiological agent of a unique chronic active hepatitis in mice. natural infection appeared to be acquired early in life in enzootically infected colonies. liver lesions arose as focal necrosis and focal nonsuppurative inflammation by 1 to 4 months of age in susceptible mouse strains. by 6 to 8 months, extensive liver involvement included ... | 1994 | 7943185 |
| 4th seah cheng siang memorial lecture. primary gastrointestinal lymphoma in hong kong. | many primary gastrointestinal lymphomas (pgl) are treatable and carry a good prognosis, particularly of localized and of "low grade" malignancy. in general, the stomach is the most commonly involved site followed by the small and then large intestines. the incidence of gastric lymphoma may be on the rise, and the response of lymphoma arising from gastric mucosa associated lymphoid tissue (malt) to treatment of the co-existing helicobacter pylori infection has added a new dimension to the study o ... | 1994 | 7944264 |
| nutritional requirements for growth of helicobacter pylori. | a chemically defined medium consisting of a buffered mineral base supplemented with amino acids, a purine, and thiamine supported growth of 23 clinical isolates and the type strain of helicobacter pylori. the growth of four strains was inhibited by the presence of certain amino acids. all but one strain required alanine for growth. the amino acids leucine, valine, phenylalanine, methionine, arginine, and histidine were generally required. isoleucine either was required or stimulated growth. stra ... | 1994 | 7944377 |
| [prevalence of helicobacter pylori infection in symptomatic adults]. | to study the prevalence of helicobacter pylori infection in different gastroduodenal entities in a wide group of symptomatic patients of our area. | 1994 | 7946600 |
| helicobacter pylori prevalence in endoscopy and medical staff. | the epidemiology and mode of transmission of helicobacter pylori is currently unclear; it is postulated that the human stomach is the natural reservoir and that spread occurs by faecal-oral or oral-oral transmission. the aim of this study was to assess the prevalence of h. pylori in gastroenterologists and gastroenterology nurses compared with internists, general nurses and the normal population. an enzyme-linked immunosorbent assay technique (sensitivity 96%, specificity 88%) was used to detect ... | 1994 | 7948812 |
| treatment of peptic ulcers from now to the millennium. | the present strategies for the management of peptic ulceration are well tolerated and clinically effective. histamine h2-receptor antagonists can be used for mild to moderate disease, and proton pump inhibitors are of particular benefit for patients with severe peptic ulceration and the zollinger-ellison syndrome. however, none of these treatments provides protection against recurrent ulceration, except when taken as long-term continuous treatment. long-term exposure to pharmacological agents ra ... | 1994 | 7949462 |
| [clinical observation on verrucous gastritis with combined therapy of traditional chinese and western medicine]. | controlled study of verrucous gastritis treated with combined therapy of tcm-wm was compared with that of wm only. the result showed that the total effective rate and the cured rate of former were 97.83% and 84.78%, while that of latter were 77.14% and 22.86% respectively. the tcm-wm revealed better results than wm alone. follow-up was done after six months, the recurrence rates of the two groups were 9.10% (2/22) and 43.75% (7/16) respectively, the difference was significant (p < 0.05). there w ... | 1994 | 7950183 |
| helicobacter pylori induces inflammation in mouse urinary bladder and pelvis. | helicobacter pylori was transurethrally inoculated into the mouse urinary tract. the organism established infection and induced inflammation in the urinary bladder and pelvis. during the infection, urinary ph was elevated, probably due to the production of nh3 by bacterial urease. h. pylori was recovered from the urinary bladder, kidney and urine of the infected mice. histopathologically, severe neutrophil infiltration was observed in the mucosal layer of both organs. h. pylori was detected on t ... | 1994 | 7935056 |
| heat shock protein produced by helicobacter pylori. | the cells of helicobacter pylori were suspended in the medium containing 35s-methionine. after a heat shock of the cells at 42 c for 5, 10, and 30 min, the production of proteins was analyzed by sodium dodecyl sulfate-polyacrylamide gel electrophoresis and autoradiography. out of many proteins produced by the cells, only 66 kda protein production was dramatically increased by heat treatment. the n-terminal amino acid sequence of 66 kda protein was quite similar to that of 62 kda and 54 kda prote ... | 1994 | 7935068 |
| evaluation of ribotyping techniques as applied to arcobacter, campylobacter and helicobacter. | restriction fragment length polymorphisms of ribosomal dna (ribotyping) of many bacterial species has been useful for both epidemiologic subtyping and species identification. however, the use of ribotyping has been confined to major research and reference laboratories due to two factors: (a) the procedure must be carefully optimized for each organism one wishes to investigate and (b) most currently available protocols use hazardous chemicals or radioisotopes. the purpose of this study is to sugg ... | 1994 | 7935508 |
| [helicobacter pylori. from controversial accidental finding to accepted pathogen]. | 1994 | 7937014 | |
| [helicobacter pylori and gastric ulcer]. | in connection with longitudinal ulcer studies and the demonstration of helicobacter pylori as the main cause of chronic gastritis, new aspects of gastric ulcer recurrences and healing become evident. this extends the possibilities to prognosticate the course of gastric ulcer and to use more effective treatment. | 1994 | 7937016 |
| [helicobacter pylori infection in children]. | approximately 10 per cent of all schoolchildren have recurrent stomach pain, and 50 per cent of those referred to hospital for gastralgia can be expected to have persistent problems as adults. some of these children might be helped by diagnosis and treatment. the lack of large controlled studies of the importance of h pylori in childhood needs to be remedied, with a view to diagnosis and treatment in the future. | 1994 | 7937017 |
| gastroenterology--i: gastroduodenal disease and helicobacter pylori. | 1994 | 7937449 | |
| cellular vacuoles induced by helicobacter pylori originate from late endosomal compartments. | pathogenic strains of helicobacter pylori cause progressive vacuolation and death of epithelial cells. to identify the nature of vacuoles, the distribution of markers of various membrane traffic compartments was studied. vacuoles derive from the endocytic pathway since they include the fluid-phase marker lucifer yellow. early endosome markers such as rab5, transferrin, and transferrin receptor, as well as the lysosomal hydrolase cathepsin d, are excluded from these structures. in contrast, the v ... | 1994 | 7937879 |
| helicobacter pylori eradication in patients with peptic ulcer disease: clinical consequences and financial implications. | we assessed clinical consequences and financial implications of helicobacter pylori eradication in 175 patients with peptic ulceration, of whom 106 had been free from h. pylori infection for a mean of 3.2 years, while 69 remained infected. we used quarterly questionnaires to examine consumption of ulcer-healing medication and antacids. in the 106 successfully treated patients, gastrointestinal haemorrhage as a complication of peptic ulcer complications during the 344 patient years after eradicat ... | 1994 | 7938408 |
| [helicobacter pylori infection and the risk of gastric carcinoma. report of a case]. | the authors present the clinical history of a patient with gastritis of the antrum who subsequently had early gastric cancer of the antrum. the authors review the literature on the infection by helicobacter pylori as a risk factor for gastric carcinoma. the infection produces chronic gastritis, gastric ulcer, duodenal ulcer and is a probable risk factor for gastric carcinoma. | 1994 | 7938716 |
| [helicobacter pylori and gastroduodenal disease]. | gastroduodenal infection by helicobacter pylori is a known cause of many gastric and duodenal disorders. infection by h. pylori is very frequent ant its prevalence increases with age by about 1% per year. human-to-human transmission appears probable. h. pylori lives under the mucous layer of gastric-type epithelium. it is the main causal agent of chronic diffuse superficial gastritis (type b). after several decades lesions of superficial gastritis can evolve to atrophic gastritis. spontaneous sh ... | 1994 | 7939299 |
| [gastritis. current point of view of the anatomo-pathologist]. | acute, granulomatous and chronic are the three main forms of gastritis. the discovery of helicobacter pylori has stimulated a new detailed classification of gastritis. having set out the various conventional histopathological classifications, the authors describe two newly recognized types of gastritis, lymphocytic gastritis and reactive gastritis. the authors also recall the earlier pathogenic classifications of gastritis, first initiated by strickland and mackay. because of the problems relate ... | 1994 | 7939325 |
| prevalence of intestinal metaplasia in helicobacter pylori gastritis. | the prevalence of intestinal metaplasia (im) in underlying helicobacter pylori gastritis was studied in 1446 patients. | 1994 | 7939396 |
| impact of metronidazole resistance on the eradication of helicobacter pylori. | we investigated how often helicobacter pylori was resistant to metronidazole before treatment in 283 h. pylori positive patients and whether the in vitro susceptibility to metronidazole could predict the clinical outcome of several drug regimens containing metronidazole. metronidazole susceptibility was tested using either disc diffusion or plates containing metronidazole. metronidazole-resistant strains were found in 41% of patients before their first anti-h. pylori treatment course. in patient ... | 1994 | 7939433 |
| [etiology, diagnosis and course of infectious diarrhea in the liestal canton hospital (5-year retrospective study)]. | between 1987 and 1991 219 patients (1.3% of all hospitalized patients) with acute infectious diarrhea were investigated retrospectively. 52% of the patients were hospitalized and 48% were outpatients. in 55% the inducing diarrhea microorganism could be identified. the most frequently detected pathogens were endemic salmonella sp. and campylobacter jejuni (65%). imported diarrheas (shigella sp. and parasites) were rare, as only 15% of the patients had a history of travel. all clostridium difficil ... | 1994 | 7939515 |
| [primary non-hodgkin lymphoma of the stomach. a review with special reference to the malt concept]. | primary extranodal malignant lymphomas are most often localized in the stomach. in contrast to gastric carcinomas, primary gastric non-hodgkin's lymphomas show an increasing incidence. according to their grade of malignancy they are divided into low-grade and high-grade non-hodgkin's lymphomas and according to their immunophenotype into b-cell and t-cell non-hodgkin's lymphomas. in most cases they show a b-cell phenotype while high-grade tumors are more frequent than those of low-grade malignanc ... | 1994 | 7939542 |
| [low-grade malt-type non-hodgkin lymphoma of the stomach with local recurrence 14 years following resection. demonstration of clonal identity using polymerase-chain-reaction (pcr)]. | so-called low grade b-cell lymphomas of malt type occurring mainly in the gastrointestinal tract but also in the salivary glands, the thyroid and the lungs are indolent neoplasms with a prolonged clinical course and persistent localized disease at the site of origin. this behaviour sets them apart from their nodal counterparts, which are frequently generalized from onset. their recognition within and their separation from accompanying reactive processes (e.g. chronic gastritis) is important but ... | 1994 | 7939543 |
| the rapid urease test in the diagnosis of helicobacter pylori infection. | with the increasing recognition of the importance of h. pylori in gastrointestinal disease, there is a need for a reliable, efficient and yet inexpensive diagnostic test. the performance of the rapid urease test (rut) as an endoscopy suite diagnostic test was compared to the established methods of culture, histology and gram stain of tissue smear, in 274 gastric biopsy samples. histology had the highest sensitivity of 99.3% followed by the rut (96.6%). culture and gram stain of tissue smear had ... | 1994 | 7939811 |
| gastrin and somatostatin in helicobacter pylori infected antral mucosa. | helicobacter pylori infection is associated with increased meal stimulated gastrin secretion, but the reason for this is unknown. sequence specific radioimmunoassays were used to measure the concentration of alpha-amidated gastrin, the total progastrin product, and somatostatin in biopsy specimens of human antral mucosa. the antral concentrations of alpha-amidated gastrin and of total progastrin products were significantly higher in h pylori infected patients than in those not infected by this o ... | 1994 | 7911115 |
| antibiotic treatment for low-grade gastric malt lymphoma. | 1994 | 7911201 | |
| antibiotic treatment for low-grade gastric malt lymphoma. | 1994 | 7911202 | |
| [active chronic gastritis and helicobacter pylori]. | biopsies from fundal and pyloric regions of the stomach of 140 patients with histologic signs of active chronic gastritis (acg) are studied. two types of acg are distinguished: associated with hp and hp-negative. inflammatory reaction in the foveal epithelium and lamina propria of the mucous membrane is characteristic for the acg of the former type. the latter is characterized by infiltration with polymorphonuclear leucocytes of the surface epithelium with its degenerative changes. in the course ... | 1994 | 7911299 |
| duodenal ulcer treated with helicobacter pylori eradication: seven-year follow-up. | the long-term benefits of helicobacter pylori-eradication treatment (het) in h pylori-associated duodenal ulcer are unclear. we followed up patients with duodenal ulcers from a trial of h pylori eradication in 1985-86. 63 of 78 patients (81%) were reviewed clinically and had upper gastrointestinal endoscopy with gastric antral biopsy. of 35 patients previously rendered h pylori negative, 32 (92%) remained h pylori negative after 7.1 years (mean). all patients initially h pylori positive remained ... | 1994 | 7905095 |
| h pylori, gastric carcinoma, and malt lymphoma. | 1994 | 7905571 | |
| h pylori and gastric cancer. | 1994 | 7904707 | |
| low dose, short duration therapy for the eradication of helicobacter pylori in patients with duodenal ulcer. | to study the efficacy and acceptability of low dose, short duration triple therapy in eradicating helicobacter pylori from patients with duodenal ulcer. | 1994 | 7906009 |
| duodenal ulcer healing by eradication of helicobacter pylori without anti-acid treatment: randomised controlled trial. | randomised trials have shown that duodenal ulcers treated by h2 blockers heal faster if helicobacter pylori is eradicated concurrently. it remains unknown whether eradication of h pylori without suppression of acid-secretion, is sufficient to allow healing. 153 patients with h pylori infection and duodenal ulcer were randomised to receive either a 1-week course of bismuth subcitrate, tetracycline, and metronidazole (76), or omeprazole for 4 weeks with the same three-drug regimen for the first we ... | 1994 | 7906759 |
| contribution of hla-dqa gene to host's response against helicobacter pylori. | 1994 | 7906783 | |
| blood group antigens and helicobacter pylori infections. | 1994 | 7906784 | |
| molecular typing of helicobacter pylori isolates from asymptomatic, ulcer and gastritis patients by urease gene polymorphism. | the gastric-adapted bacterium helicobacter pylori plays an important role in gastritis and ulcer disease, but no phenotypic typing scheme presently exists for this organism. with a view to the development of genotypic typing, we have compared isolates of h. pylori from gastritis or ulcer patients with those from subjects exhibiting no disease. variation was analysed at the urease genes, urea and urecd, by employing pcr-generated probes in genomic southern blot hybridizations. whilst urea restric ... | 1994 | 7907028 |
| comparison of labelling by bromodeoxyuridine, mib-1, and proliferating cell nuclear antigen in gastric mucosal biopsy specimens. | to compare proliferating cell nuclear antigen (pcna) and mib-1 with bromodeoxyuridine (brdu) pulse labelling, a specific marker of cell proliferation, in endoscopic gastric biopsy specimens. | 1994 | 7907613 |
| helicobacter pylori eradication. | 1994 | 7908097 | |
| therapeutic immunisation against helicobacter infection. | 1994 | 7908372 | |
| duodenal ulcer healing drugs. | 1994 | 7908373 | |
| duodenal ulcer healing drugs. | 1994 | 7908374 | |
| duodenal ulcer healing drugs. | 1994 | 7908375 | |
| [somatostatin--the decisive connection between helicobacter pylori gastritis and duodenal ulcer?]. | 1994 | 7908475 | |
| pcr-based restriction fragment length polymorphism typing of helicobacter pylori. | we applied a molecular typing approach for helicobacter pylori that uses restriction fragment length polymorphism (rflp) analyses of an 820-bp pcr-amplified portion of the urec gene in h. pylori. the pcr products were digested with restriction enzyme hhai, mboi, or msei, and the fragments generated were analyzed by agarose electrophoresis. among 25 independent clinical isolates, each showed a different pattern when a combination of the three rflp patterns was used. using this method, we studied ... | 1994 | 7908671 |