Publications
| Title | Abstract | Year Filter | PMID(sorted ascending) Filter |
|---|
| isolation from gastric epithelium of campylobacter-like bacteria that are distinct from "campylobacter pyloridis". | 1985 | 2857011 | |
| immune response to campylobacter pyloridis in patients with peptic ulceration. | 1985 | 2858757 | |
| immune response to campylobacter pyloridis. | 1985 | 2860417 | |
| campylobacter pyloridis in peptic ulcer. | 1985 | 2861380 | |
| immunological identification of campylobacter pyloridis in gastric biopsy tissue. | 1985 | 2861476 | |
| campylobacter pyloridis is unique: gclo-2 is an ordinary campylobacter. | 1985 | 2861477 | |
| rapid diagnosis of campylobacter pyloridis infection. | 1986 | 2867361 | |
| campylobacter pyloridis and gastritis in children. | 1986 | 2868325 | |
| rapid diagnosis of campylobacter pyloridis gastritis. | 1986 | 2868326 | |
| campylobacter pyloridis in children. | 1986 | 2870240 | |
| urea hydrolysis in patients with campylobacter pyloridis infection. | 1986 | 2871253 | |
| campylobacter pyloridis, urease, hydrogen ion back diffusion, and gastric ulcers. | campylobacter pyloridis, a bacterium implicated as the aetiological agent of gastritis and possibly gastric ulcers, has a very high urease activity. the rapid hydrolysis of urea at intercellular junctions results in alterations in the milieu of the gastric epithelium preventing the normal passage of hydrogen ions (h+) from the gastric glands through the mucus to the lumen and permits back diffusion. a consequence of h+ back diffusion is hypochlorhydria and a predisposition to ulcer formation. se ... | 1986 | 2873317 |
| campylobacter pyloridis, urease, and gastric ulcers. | 1986 | 2874399 | |
| campylobacter pyloridis, urease, and gastric ulcers. | 1986 | 2875334 | |
| in-vitro sensitivity of campylobacter pyloridis to furazolidone. | 1986 | 2877192 | |
| effect of duodenal ulcer surgery and enterogastric reflux on campylobacter pyloridis. | to assess the effect of duodenal ulcer surgery on campylobacter pyloridis gastric biopsies were done and fasting bile acid concentrations in gastric aspirates were measured in 35 patients with active duodenal ulceration and 54 who had undergone surgery at some time. biopsy specimens were assessed blind for the presence of c pyloridis and scored for severity of reflux gastritis by the use of a histological grading system. among patients who had undergone highly selective vagotomy the proportion w ... | 1986 | 2877324 |
| campylobacter pyloridis infection as possible complication of weight loss therapy. | 1986 | 2878218 | |
| monoclonal antibodies for rapid identification of campylobacter pyloridis. | 1986 | 2878265 | |
| treatment failure of ofloxacin in campylobacter pylori infection. | 1987 | 2883434 | |
| serological screening for campylobacter pylori in candidates for renal transplantation. | 1987 | 2883460 | |
| campylobacter pylori detected noninvasively by the 13c-urea breath test. | the high endogenous urease activity of campylobacter pylori was exploited in a non-invasive test for the presence of this organism in the stomach. when 13c-urea was administered orally after a test meal, urea-derived 13co2 appeared in the respiratory co2 of infected individuals at a constant rate for greater than 100 min. the test was validated in 26 individuals who underwent both the 13c-urea breath test and endoscopic biopsy of the antral mucosa for culture and histological examination. each p ... | 1987 | 2883491 |
| 14c-urea breath analysis, a non-invasive test for campylobacter pylori in the stomach. | 1987 | 2884468 | |
| campylobacter pylori infection in homosexuals. | 1987 | 2885612 | |
| campylobacter pylori and peptic ulcer. | 1987 | 2886854 | |
| campylobacter pylori and peptic ulcer disease. | 1987 | 2887926 | |
| person-to-person transmission of campylobacter pylori. | 1987 | 2887955 | |
| antibody to campylobacter pylori in families of index children with gastrointestinal illness due to c pylori. | 1987 | 2887956 | |
| campylobacter pylori and gastric manipulation for morbid obesity. | 1987 | 2889113 | |
| campylobacter pylori and recurrence of duodenal ulcers--a 12-month follow-up study. | in 39 patients with endoscopically healed duodenal ulcers repeat endoscopy and two antral biopsies after 1 year showed a relapse rate of 59%. only post-treatment campylobacter pylori status was a significant predictor of endoscopic relapse. 79% of patients who remained culture positive had a relapse, compared with 27% of culture-negative patients. relapse was more likely (66%) in patients with a recurrence of c pylori after apparent eradication of the organism than in those who remained negative ... | 1987 | 2890019 |
| naturally occurring gastritis associated with campylobacter pylori infection in the rhesus monkey. | 1987 | 2890941 | |
| campylobacter pylori and pernicious anaemia. | 1988 | 2891919 | |
| campylobacter pylori and duodenogastric reflux in peptic ulcer disease and gastritis. | 1988 | 2891956 | |
| campylobacter pylori and development of duodenal ulcer. | 1988 | 2891957 | |
| a decade of campylobacter pylori. | 1988 | 2893219 | |
| campylobacter pylori and ulcer recurrence. | 1988 | 2894530 | |
| one-minute endoscopy room test for campylobacter pylori. | 1988 | 2895232 | |
| campylobacter pylori and protein losing enteropathy in children. | 1988 | 2895370 | |
| campylobacter pylori urease: a new serological test. | 1988 | 2896815 | |
| campylobacter pylori in patients with gastritis, peptic ulcer, and carcinoma of the stomach in lanzhou, china. | 1988 | 2896897 | |
| [ofloxacin in therapy of "resistant" duodenal ulcer. a pilot study]. | since its description in 1983 campylobacter pylori (c.p.) has been discussed as possible pathogenic factor at least in duodenal ulcer disease. the therapeutic combination of h2-receptor-antagonists (= h2-ra) and ofloxacin (tarivid) has shown to heal resistant duodenal ulcers in some preliminary cases, which did not respond to a three month standard dosage treatment with h2-ra. this paper describes the results of twelve patients whose duodenal ulcers were resistant to h2-ra. c.p. was detected in ... | 1988 | 2900584 |
| campylobacter pylori in abattoir workers: is it a zoonosis? | sera from 98 abattoir workers were tested for igg to campylobacter pylori, c jejuni, and klebsiella. clerical workers had significantly lower c pylori and c jejuni igg titres than any of the groups in direct contact with freshly cut animal parts. no difference was found for antibodies to klebsiella. 28 non-clerical workers with high-titre c pylori igg consented to upper gastrointestinal endoscopy. c pylori associated gastritis was found in all 28, and four weeks of colloidal bismuth subcitrate ( ... | 1988 | 2901576 |
| effect of acid inhibition on campylobacter pylori. | campylobacter pylori (cp) infection has been demonstrated in 59 of 65 (91%) patients with gastric or duodenal ulcer. patients were included in a double blind study with ranitidine or famotidine for 4-8 weeks. after therapy, all gastric and duodenal ulcers healed but cp remained in all previously infected patients, associated with chronic inflammation of gastric mucosa. our results suggest that h2 antagonists do not have any effect either on cp infection or on concurrent chronic gastritis. at the ... | 1988 | 2901784 |
| is campylobacter pylori a zoonosis? | 1988 | 2902418 | |
| is campylobacter pylori a zoonosis? | 1988 | 2903369 | |
| the pathophysiological and pharmacological basis of peptic ulcer therapy. | both genetic and nongenetic factors predispose to ulcer diathesis. at the mucosal level ulcers result from an imbalance between aggressive factors and mucosal defense. ulcer therapy reduces aggressive forces, bolsters defense, or both. gastric acid, the major aggressive factor, may have its secretion inhibited or it may be partially neutralized by antacids. h2 receptor antagonists competitively block histamine occupancy of h2 receptors on parietal cells, thereby preventing stimulation of adenyla ... | 1988 | 2903542 |
| "eradication" of campylobacter pylori: are we being misled? | 1988 | 2903967 | |
| prospective double-blind trial of duodenal ulcer relapse after eradication of campylobacter pylori. | 100 consecutive patients with both duodenal ulcer and campylobacter pylori infection were followed up to see whether eradication of c pylori affected ulcer healing or relapse. patients were randomly assigned to 8 weeks of treatment with cimetidine or colloidal bismuth subcitrate (cbs), with tinidazole or placebo being given concurrently from days 1 to 10, inclusive. endoscopy, biopsy, and culture were done at entry, in weeks 10, 22, 34, and 62, and whenever symptoms recurred. there was no mainte ... | 1988 | 2904568 |
| duodenal ulcer, campylobacter pylori, and the "leaking roof" concept. | 1988 | 2904580 | |
| future trends in the management of peptic ulcer disease. | future trends in the short- and long-term management of peptic ulcer disease are considered. the present state of development of pharmacologic agents for the short-term healing of duodenal and gastric ulcer is impressive, and high rates of healing with rapid symptom relief can be safely achieved with several agents. the problem of ulcer recurrence has not been solved, although new work concerning the role of campylobacter pylori holds promise. the mortality of acute gastrointestinal haemorrhage ... | 1988 | 2906462 |
| comparison between medical and elective surgical treatment of peptic ulcers. | during the last decades we have learnt how to treat chronic peptic ulcer disease both with surgery (mainly vagotomy) and with long term medical treatment (mainly h2-receptor antagonists) with great success and safety. although much attention has lately been given to 'cytoprotective' agents, it is still too early to regard them as alternatives to acid reducing treatment in the long term management of severe peptic ulcer disease. the importance of campylobacter pylori for long term outcome is toda ... | 1988 | 2907681 |
| cytotoxin production by campylobacter pylori strains isolated from patients with peptic ulcers and from patients with chronic gastritis only. | a total of 66.6% of campylobacter pylori strains isolated from patients with peptic ulcers produced a cytotoxin active against mammalian cells in vitro, versus 30.1% of strains isolated from patients with chronic gastritis of various degrees of severity only. this difference was statistically significant and suggests that the toxic substance could be involved in the development of peptic ulcers. | 1989 | 2913034 |
| detection of antibody responses in rabbits hyperimmunized with campylobacter pylori. enzyme immunoassay indicates extensive antigenic similarities. | acid glycine extracts from four campylobacter pylori strains and one gclo strain were used as antigens in enzyme immunoassay (eia). immune responses of rabbits immunized with c. pylori strains, the gclo-strain and other campylobacter strains were studied. all 14 rabbit antisera against c. pylori reacted with all four c. pylori extracts and there were extensive cross-reactions between these extracts. antisera against c. jejuni and c. coli strains did not react with a c. pylori extract but reached ... | 1989 | 2914106 |
| identification of campylobacter pylori by using the rapid nh system. | campylobacter pylori has been associated with chronic active antral gastritis. the organism was isolated from 19 of 45 gastric mucosal biopsies on blood agar plates with increased co2 at 35 degrees c. the rapid nh system, a set of dehydrated substrates for preformed enzymes, was used to assist in the identification of c. pylori. all c. pylori gave the same biochemical profile, and it was different from those of all other organisms in the profile index of the manufacturer. the rapid nh system is ... | 1989 | 2915028 |
| serum igg antibody to the outer membrane proteins of campylobacter pylori in children with gastroduodenal disease. | 1989 | 2915172 | |
| usefulness of culture, histology, and urease testing in the detection of campylobacter pylori. | several tests have been described which detect the presence of campylobacter pylori (cp) in the human upper gastrointestinal tract. antral biopsies from 160 endoscopies were evaluated for the presence of cp; four tests readily available to the practicing endoscopist were used: two urease tests [clotest and microtiter biopsy urease test (mbut)], hematoxylin and eosin staining, and culture. sensitivity and specificity of each was evaluated and found to be near 90%, except for culture, which was le ... | 1989 | 2916521 |
| campylobacter pylori and gastric acidity. | 1989 | 2916534 | |
| one-minute test for campylobacter pylori. | 1989 | 2916535 | |
| [modern gastritis therapy. new pathogenetic concepts. significance of campylobacter pylori. use of bismuth compounds. report of the 16th meeting of the bayern society of gastroenterology. bad dürkheim, 21-22 october 1988]. | 1989 | 2917722 | |
| serum antibody responses to the n-acetylneuraminyllactose-binding hemagglutinin of campylobacter pylori. | we recently reported that campylobacter pylori possesses a surface-associated fibrillar hemagglutinin which has an affinity for n-acetylneuraminyllactose; this hemagglutinin may function as a colonization factor for attachment to the gastric epithelium. in the present study we examined serum samples obtained from 65 c. pylori-infected individuals with gastric ulcers, duodenal ulcers, or both and from 121 asymptomatic volunteers, including 62 who were infected with c. pylori, for immunoglobulin g ... | 1989 | 2917777 |
| hemagglutination activity of campylobacter pylori. | forty-five strains of campylobacter pylori isolated from gastric biopsy specimens showed distinct hemagglutination activity. the activity was partially decreased by treatment with heat, trypsin, or an alkylating agent and was inhibited by porcine gastric mucin but not by various compounds, including d-mannose. | 1989 | 2917797 |
| [campylobacter pylori--an amazing discovery still of current interest]. | 1989 | 2918773 | |
| in vivo susceptibility of campylobacter pylori. | campylobacter pylori infection has been associated with duodenal ulcer, gastric ulcer, and non-ulcer dyspepsia. although in vitro studies have shown that c. pylori is susceptible to most commonly used antibiotics, predictions from in vitro sensitivity studies have not led to a safe and generally effective therapy; c. pylori has proved to be very difficult to eradicate in vivo. we used the urea breath test to assess the susceptibility of c. pylori in vivo to various drugs. c. pylori was susceptib ... | 1989 | 2919580 |
| campylobacter pylori in esophagus, antrum, and duodenum. a histological and microbiological study. | two hundred forty-six patients with a wide range of upper gastrointestinal tract disorders were investigated for the presence of campylobacter pylori infection in esophagus, gastric antrum, and duodenum. c. pylori was identified in 52% of patients in at least one site, and microbiological and histological techniques were used to exclude the presence of the organism. esophageal infection was not significant and is probably due to reflux. antral c. pylori was significantly associated with active g ... | 1989 | 2920650 |
| campylobacter-like organisms and heterotopic gastric mucosa in meckel's diverticula. | to assess the possibility that campylobacter pylori might colonise heterotopic gastric mucosa a detailed histological review of 69 cases of meckel's diverticula resected over 17 years was undertaken. twenty three were resected incidentally while 46 were excised as the suspected cause of symptoms. gastric mucosa was found in 13 diverticula (19%), 10 from the symptomatic group and three from the incidental cases, of which eight showed active gastritis affecting the heterotopic mucosa. specific sta ... | 1989 | 2921353 |
| sensitivity of campylobacter pylori to colloidal bismuth subcitrate. | 1989 | 2921366 | |
| [campylobacter pylori and gastritis. association or induction?]. | 1989 | 2922375 | |
| [morphologic change in campylobacter pylori associated gastritis in and following successful antibacterial therapy]. | 1989 | 2922381 | |
| campylobacter pylori, hypertrophic erosive gastritis and hypoalbuminemia healed by cephalexin therapy. | 1989 | 2925040 | |
| a sensitive and specific serologic test for detection of campylobacter pylori infection. | campylobacter pylori has been associated with gastritis, duodenal ulcer, gastric ulcer, and nonulcer dyspepsia. evidence that c. pylori may be the causative agent or at least a major contributory factor in peptic ulcer disease has generated intense interest in the development of reliable methods for detecting c. pylori infections. we have developed a specific and sensitive enzyme-linked immunosorbent assay (elisa) that detects serum immunoglobulin g antibodies directed against high molecular wei ... | 1989 | 2925047 |
| campylobacter pylori and symptoms: is there a cause and effect relationship? | 1989 | 2925068 | |
| campylobacter pylori in acquired immunodeficiency syndrome. | 1989 | 2925069 | |
| campylobacter pylori virulence factors in gnotobiotic piglets. | thirty-three gnotobiotic piglets from four litters were challenged with motile and nonmotile strains of campylobacter pylori. the most motile strain, 26695, was the most virulent, with a 100% infection rate. the least motile strain, tx30a, was the least virulent, with an infection rate of only 17%. strain 60190 was weakly motile and had intermediate virulence, with an infection rate of 40%. strains recovered from piglets were more motile than the challenge strains. the challenge strains also dif ... | 1989 | 2925243 |
| susceptibility of clinical isolates of campylobacter pyloridis to 11 antimicrobial agents. | the activities of 11 antimicrobial agents, including two bismuth salts, against 70 strains of campylobacter pyloridis isolated from gastric biopsy specimens were tested. the isolates were very susceptible to penicillin (the mic for 90% of the strains tested [mic90] was 0.03 microgram/ml), erythromycin, cefoxitin (mic90, 0.12 microgram/ml), gentamicin, and ciprofloxacin (mic90, 0.25 microgram/ml). the bismuth salts and nalidixic acid had moderate activity (mic90, 16 to 64 micrograms/ml). twenty p ... | 1985 | 2935076 |
| [isolation of campylobacter pylori from antropyloric biopsies. a year of systematic research]. | 1988 | 2965362 | |
| [initial experience with the detection of campylobacter pylori]. | 1988 | 2967116 | |
| [campylobacter pyloridis and gastroduodenal inflammation]. | 1988 | 2972404 | |
| [do nonsteroidal anti-inflammatory drugs have a protective effect against campylobacter pylori?]. | 1988 | 2978311 | |
| campylobacter pylori-associated gastritis: attempts to eradicate the bacteria by various antibiotics and anti-ulcer regimens. | the efficacy of various antimicrobial and anti-ulcer agents on the eradication of campylobacter pylori in patients with antral gastritis or duodenal ulcers was investigated by several open studies or double-blind, placebo-controlled protocols. among the anti-ulcer agents, ranitidine, cimetidine or sucraflate had no effect on c. pylori. colloidal bismuth subcitrate achieved clearance of c. pylori in 40% of treated patients at the end of therapy but a high relapse rate (14/16 patients) was observe ... | 1988 | 2979039 |
| is it really more difficult to treat prepyloric ulcers? | prepyloric and duodenal ulcers have some common characteristics: gastric acid secretion is increased and there is an association with blood group o. many, therefore, have considered prepyloric ulcers to be a variety of duodenal ulcer disease. from an anatomical point of view, however, prepyloric ulcers are clearly gastric ulcers. after proximal selective vagotomy, the recurrence rate is very high, amounting to more than 30% in 5 years; this is significantly higher than the rate for duodenal ulce ... | 1987 | 2979694 |
| what is new in the epidemiology and pathogenesis of peptic ulcer? | the old dictum 'no acid--no ulcer' is no longer a sufficient explanation of the pathogenesis of ulcer disease. the real question is 'if acid--why ulcer?' although acid remains predominant, some of the other factors influencing ulcerogenesis are nocturnal acid secretion, pepsin enzyme subspecies, the mucus layer, bicarbonate levels, prostaglandins, campylobacter pylori infection, consumption of non-steroidal anti-inflammatory drugs, and smoking habits. although the ulcer burden has been greatly r ... | 1987 | 2979699 |
| significance of campylobacter pylori. | there is an explosion of interest in the role of campylobacter pylori as a cause of active chronic gastritis. this curved spiraled microorganism can readily be detected within the mucus gel covering the stomach mucosa, especially in patients suffering from peptic ulcer disease or non-ulcer dyspepsia. to what extent this intriguing microorganism is causally related to peptic ulcer disease remains to be elucidated, but all the evidence which is available so far supports a pathogenetically importan ... | 1987 | 2979701 |
| problems with clinical trials on campylobacter pylori. | clinical trials investigating campylobacter pylori infection have proven difficult to conduct and to interpret. those trials examining the relapse rates of duodenal ulcer disease among subjects in whom c. pylori infection has been eradicated have shown a significant reduction in recurrence rates. however, since the bismuthate compounds used in these trials have cytoprotective ulcer-healing properties, reduced relapse rates cannot be attributed to antibiotic activity alone. those trials examining ... | 1988 | 2980756 |
| identification of campylobacter pyloridis isolates by restriction endonuclease dna analysis. | campylobacter pyloridis isolates recovered from gastric biopsy specimens of 16 patients were examined by restriction endonuclease dna analysis with hindiii. for 8 of these 16 patients two different isolates were compared to study the persistence of the colonizing strains and the stability of their dna digest patterns during a period of 2 years (two patients), the identity or nonidentity of different colony types within one culture (two patients), and the nature of the relapses after apparently s ... | 1986 | 3020084 |
| [effect of bismuth subsalicylate versus cimetidine on campylobacter pylori, ulcer healing and rate of recurrence]. | 1988 | 3043160 | |
| [campylobacter pyloridis and diseases of the stomach and duodenum]. | 1988 | 3046137 | |
| monoclonal antibodies for detection of campylobacter pylori in biopsy smears and frozen sections. | 1988 | 3047850 | |
| heterogeneity of campylobacter pylori as demonstrated by co-agglutination testing with rabbit antibodies. | the indirect immunofluorescence (ifl) and the co-agglutination (coa) methods were used to study the serology of campylobacter pylori strains isolated from patients in different countries (sweden, finland, canada and australia). antisera were obtained from rabbits immunized with whole cell antigens. with ifl tests the highest serum titers were obtained with c. pylori strains and their homologous antisera. these tests also showed that all the tested strains contained cross-reactive antigens. with ... | 1988 | 3047851 |
| comparison of different tests for campylobacter pylori. | 1988 | 3047852 | |
| bismuth: effects on gastritis and peptic ulcer. | the healing properties of colloidal bismuth subcitrate (cbs) on peptic ulcer are well established and several studies have shown that healing with cbs is associated with a lower relapse rate than that produced by h2-receptor antagonists. the recent observation that cbs is effective against campylobacter pylori has shed light on this because recent studies have shown that eradication of c. pylori by cbs leads to resolution of the associated gastritis and this may explain the low relapse rates. cb ... | 1988 | 3047853 |
| the role of antibiotics in campylobacter pylori associated peptic ulcer disease. | since the recognition and first culture of campylobacter pylori (cp) the hypothesis of a pathogenic role has been strengthened by numerous investigations. its close association with active chronic gastritis and even active duodenitis, and the disappearance of these pathologic conditions with antimicrobial treatment suggest a pathogenic role for cp in active chronic gastritis. the close association of antral gastritis with duodenal ulcer (du) suggests that cp associated active chronic gastritis m ... | 1988 | 3047854 |
| gastroduodenal complications of chronic nsaid therapy. | the fact that nonsteroidal anti-inflammatory drugs (nsaids) damage the gastroduodenal mucosa is no longer contested. endoscopic studies in normal volunteers after nsaid administration have failed to predict which nsaids would be safest when administered chronically. nsaid use has been associated with a disproportionately high frequency of upper gastrointestinal bleeding and perforation of ulcers. all of the newer nsaids appear to be similar in their propensity to cause mucosal damage, including ... | 1988 | 3048080 |
| bacteria in ulcer pathogenesis. | a great deal of information about the spiral bacteria of the stomach has accumulated in the past 5 years. these bacteria, currently named campylobacter pylori but likely to be renamed as a new genus, have adapted to living beneath the mucus layer and above the gastric surface mucous cells. when metaplastic gastric mucous cells are also present in the duodenal bulb, c. pylori may also get a foothold in this latter location. observations of the high prevalence of c. pylori in patients with gastrit ... | 1988 | 3048451 |
| a review of campylobacter pylori in upper gastrointestinal disease. | the organism campylobacter pylori is frequently found in association with gastritis and peptic ulcer disease. whether it is the cause, a contributory factor or a simple commensal is not known but there is evidence to support a pathological role. current research may alter our understanding of the causes of upper gastrointestinal disease and have important implications for treatment. | 1988 | 3048506 |
| nitrofurans in the treatment of gastritis associated with campylobacter pylori. the gastrointestinal physiology working group of cayetano heredia and the johns hopkins universities. | we conducted a double-blind, placebo-controlled, randomized treatment study in peruvian adults with antral gastritis associated with campylobacter pylori. patients received either 400 mg of furazolidone (n = 14), 400 mg of nitrofurantoin (n = 24), or a placebo (n = 31) for 14 days. endoscopy was carried out at baseline, 1 day after ceasing therapy, and 6 wk after the end of treatment to verify colonization by c. pylori and determine the extent of gastric inflammation. treatment with nitrofuranto ... | 1988 | 3049213 |
| opsonophagocytosis of campylobacter pylori. | the opsonic activity of human serum from various sources against campylobacter pylori was compared. all sera, whether from control subjects with no symptoms of gastritis or peptic ulceration, or from symptomatic patients from whom c. pylori had or had not been isolated, opsonised c. pylori equally well. opsonisation depended on the alternative pathway of complement activation but not on antibody. these findings suggest that antibody plays no role in protection against c. pylori and that the pres ... | 1988 | 3050112 |
| colloidal bismuth subcitrate. a review of its pharmacodynamic and pharmacokinetic properties, and its therapeutic use in peptic ulcer disease. | colloidal bismuth subcitrate (cbs) possesses at least equal efficacy with histamine h2-receptor antagonist drugs in the treatment of peptic ulcer disease. however, cbs has the advantage of slower ulcer relapse rates than those seen after initial healing with the h2-antagonists. it has been postulated that this effect may be partly due to the antibacterial properties of cbs against campylobacter pylori, a bacterium found in the gastric mucosa and gastric metaplasia within the duodenum of most pat ... | 1988 | 3053124 |
| campylobacter pyloridis is not responsible for duodenal ulcer formation: results of a controlled therapeutic trial. | 1988 | 3053613 | |
| treatment of campylobacter pylori gastritis: a pilot study using pirenzepine dihydrochloride (gastrozepin) and three formulations of colloidal bismuth subcitrate (de-nol). | antral biopsies were obtained to detect campylobacter pylori infection in 382 patients referred for gastroscopy. one hundred and seventy four patients (46%) were infected. infection was strongly associated with histological gastritis (p less than 0.001), but there was no association between histological antral gastritis and the appearance of the gastric antrum during gastroscopy. because it has been suggested that the lower relapse rate for duodenal ulcer following colloidal bismuth subcitrate ( ... | 1988 | 3054641 |
| campylobacter pylori and gastroduodenal disease: a prospective endoscopic study and comparison of diagnostic tests. | a series of 72 adult patients undergoing 76 upper gastrointestinal (gi) endoscopies because of gi signs or symptoms were studied for the presence of campylobacter pylori by culture, histology, and rapid urease determination of gastric antral biopsy specimens. c. pylori was found by culture or histology in all 10 cases of gastric and duodenal ulcer, and in 77% of endoscopies with histologically proven active gastritis. positive culture for c. pylori was highly correlated pathologically with activ ... | 1988 | 3055942 |
| [campylobacter pylori, ulcer and gastritis: another piece from an old puzzle?]. | 1988 | 3056155 |