evidence for immunodepression of syrian hamsters and mongolian jirds by dipetalonema viteae infections. 1976557249
primary infections of dipetalonema viteae in an outbred and five inbred strains of golden hamsters. 1978565398
dipetalonema viteae infection in three species of rodents: species specific patterns of the antibody response.groups of jirds (meriones unguiculatus), multimammate rats (mastomys natalensis) and golden hamsters (mesocricetus auratus) were infected with third stage larvae (l3) of dipetaloneam viteae and the course of infection was followed until 20 weeks post infection. worm growth was best and microfilaraemia was high and long lasting in jirds and in multimammate rats, whereas golden hamsters were poor hosts as measured by these parameters. the igg and igm antibody responses of the species were compared ...19873550602
[serological cross-reactions between echinococcus multilocularis and dipetalonema viteae infected rodents (author's transl)].sera from 30 e. multilocularis infected nmri-mice and 30 d. viteae infected golden hamsters were tested in the indirect fluorescent antibody (ifat) and indirect hemagglutination (iha) tests. in the ifat there were cross-reactions with all sera, but there were differences between the average titres. while filarial antisera gave higher titres in the homologous system (1:640 against 1:80), it was the opposite with echinococcus antisera (1:160 against 1:640). other differences referred to the parasi ...19817048667
acanthocheilonema viteae: vaccination with irradiated l3 induces resistance in three species of rodents (meriones unguiculatus, mastomys coucha, mesocricetus auratus).three species of rodents were immunized with 50 irradiated (35 krad) stage-3 larvae (l3) of the filaria acanthocheilonema viteae and challenged with an infection of normal l3. the immunization induced a significant reduction of the worm burden developing from the challenge infection in all host species, the jird (meriones unguiculatus), the multimammate rat (mastomys coucha) and the golden hamster (mesocricetus auratus). the induced resistance was highest in jirds (92.5 +/- 9.7) followed by gold ...19979018308
immunocytochemical studies on several developmental stages of dipetalonema viteae (filarioidea).the binding of antibodies to infective larvae (l3), eggs, uterine contents and blood microfilariae was demonstrated by light microscopy employing the peroxidase-antiperoxidase (pap) method. antigen-antibody reactions were located on the shell of filarial eggs as well as on the cuticle and the interior of l3 and microfilariae. using sera from meriones unguiculatus, mastomys natalensis and golden hamsters infected with d. viteae obtained at 7, 14, 21 and 28 weeks p.i. it was observed that the inte ...19836685362
[effect of 17 beta-estradiol, progesterone and testosterone on dipetalonema vitae parasitosis in the castrated female golden hamster cricetus auratus].in dipetalonema viteae filariasis of cricetus auratus, it seems that the natural estrogenic hormon, the 17 beta-estradiol as well as the progesterone have a part in the protection observed in the females.19846540541
[influence of the sex of the final host on experimental dipetalonema vitae filariosis in golden hamsters cricetus auratus (author's transl)].the authors have studied the influence of the sex of the host on experimental dipetalonema viteae parasitosis in golden hamsters cricetus auratus. the parasited hamsters are sacrificed fifteen days after testing for microfilarial count, and then the extent of the infestation is measured by counting the number of male worms and female worms. the average level of microfilarial count is significantly higher in male hamsters than in female hamsters. the same applies to the extent of parasitism: the ...1979575275
studies on dipetalonema viteae (filarioidea) 3. antibody-dependent cell-mediated destruction of microfilariae in vivo.antibody-dependent cell-mediated destruction of dipetalonema viteae microfilariae could be demonstrated in the golden hamster using a micropore chamber technique. microfilariae were eliminated within 24 hours in chambers of 3.0 and 5.0 microm pore size when implanted into amicrofilaremic hamsters (week 30 post infection). at peak microfilaremia (week 12 post infection), only some hamsters could efficiently destroy microfilariae. in chambers with 0.3 microm pore size, microfilariae survived for m ...1979375512
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